Paula L Vieira-Brock
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Explore the profile of Paula L Vieira-Brock including associated specialties, affiliations and a list of published articles.
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14
Citations
258
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Recent Articles
1.
Andersen A, Vieira-Brock P, Vaughan B, Vollmer D
J Immunol Methods
. 2021 Nov;
499:113175.
PMID: 34744024
Background: Colostrum is the first milk that supplies newborns with immune supporting peptides. Due to its heterogeneous and variable characteristics, standardized assays for assessment of its biological activities are a...
2.
McFadden L, Vieira-Brock P
Toxicol Open Access
. 2019 Apr;
2(2).
PMID: 30957071
Objective: Methamphetamine (METH) is a highly addictive substance abused world-wide in both males and females. Preclinical studies in male rodents suggest that large-dose exposure to METH can lead to persistent...
3.
Vieira-Brock P, Vaughan B, Vollmer D
Biochim Open
. 2018 Feb;
5:8-13.
PMID: 29450151
Plant essential oils (EOs) are known to inhibit the growth of bacteria and fungi. Whether these antimicrobial effects are comparable to synthetic household products is less clear. Furthermore, limited research...
4.
Vieira-Brock P, McFadden L, Nielsen S, Ellis J, Walters E, Stout K, et al.
J Pharmacol Exp Ther
. 2015 Sep;
355(3):463-72.
PMID: 26391161
Repeated methamphetamine (METH) administrations cause persistent dopaminergic deficits resembling aspects of Parkinson's disease. Many METH abusers smoke cigarettes and thus self-administer nicotine; yet few studies have investigated the effects of...
5.
Vieira-Brock P, McFadden L, Nielsen S, Smith M, Hanson G, Fleckenstein A
Int J Neuropsychopharmacol
. 2015 Jul;
18(12).
PMID: 26164716
Background: Previous studies have demonstrated that methamphetamine abuse leads to memory deficits and these are associated with relapse. Furthermore, extensive evidence indicates that nicotine prevents and/or improves memory deficits in...
6.
McFadden L, Vieira-Brock P, Hanson G, Fleckenstein A
Neuropharmacology
. 2015 Feb;
93:146-54.
PMID: 25645392
Others and we have reported that prior methamphetamine (METH) exposure attenuates the persistent striatal dopaminergic deficits caused by a subsequent high-dose "binge" METH exposure. The current study investigated intermediate neurochemical...
7.
McFadden L, Vieira-Brock P, Hanson G, Fleckenstein A
Int J Neuropsychopharmacol
. 2014 Mar;
17(8):1315-20.
PMID: 24650575
Preclinical studies suggest that prior treatment with escalating doses of methamphetamine (METH) attenuates the persistent deficits in hippocampal serotonin (5-hydroxytryptamine; 5HT) transporter (SERT) function resulting from a subsequent 'binge' METH...
8.
Vieira-Brock P, Andrenyak D, Nielsen S, Fleckenstein A, Wilkins D
Nicotine Tob Res
. 2013 Jun;
15(11):1839-48.
PMID: 23737496
Introduction: Studies have evaluated the behavioral and neurochemical impact of nicotine administration in rodents. However, the distribution of nicotine and metabolites in rat brain and plasma as a function of...
9.
McFadden L, Hunt M, Vieira-Brock P, Muehle J, Nielsen S, Allen S, et al.
Drug Alcohol Depend
. 2012 Jun;
126(1-2):87-94.
PMID: 22647900
Background: Pre-clinical studies indicate that high-dose, non-contingent methamphetamine (METH) administration both rapidly and persistently decreases serotonergic neuronal function. Despite research indicating the hippocampus plays an important role in METH abuse...
10.
McFadden L, Stout K, Vieira-Brock P, Allen S, Nielsen S, Wilkins D, et al.
Synapse
. 2011 Nov;
66(3):240-5.
PMID: 22120988
Numerous preclinical studies have demonstrated that noncontingent methamphetamine (METH) administration rapidly decreases both dopamine (DA) transporter (DAT) and vesicular monoamine-2 transporter (VMAT-2) function. Because of the importance of transporter function...