P J Kuhlencordt
Overview
Explore the profile of P J Kuhlencordt including associated specialties, affiliations and a list of published articles.
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9
Citations
436
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Recent Articles
1.
Huang H, Koelle P, Fendler M, Schrottle A, Czihal M, Hoffmann U, et al.
Atherosclerosis
. 2014 May;
235(1):213-22.
PMID: 24858340
Objective: Deletion of inducible nitric oxide synthase (iNOS) in apolipoprotein E knockout mice was shown to mitigate the extent of arteriosclerosis. Oxidized low density lipoprotein (oxLDL) inhibits macrophage migration and...
2.
Czihal M, Tato F, Hoffmann U, Kuhlencordt P
Clin Exp Rheumatol
. 2013 Jan;
31(1 Suppl 75):S89.
PMID: 23294490
No abstract available.
3.
Czihal M, Zanker S, Rademacher A, Tato F, Kuhlencordt P, Schulze-Koops H, et al.
Scand J Rheumatol
. 2012 Mar;
41(3):231-6.
PMID: 22400812
Objectives: The aim of our study was to describe the sonographic pattern and clinical manifestations of extracranial (i.e. carotid and proximal arm arteries) and cranial arterial involvement in patients with...
4.
Bauersachs R, Kuhlencordt P
Dtsch Med Wochenschr
. 2011 Jan;
136(5):168-71.
PMID: 21271473
Superficial vein thrombosis (SVT) occurs at least as frequent as deep vein thrombosis (DVT), and shares common risk factors with venous thromboembolism. The CALISTO trial was the first to provide...
5.
Kuhlencordt P, Roling J, Hoffmann U
Internist (Berl)
. 2009 Jun;
50(8):945-51.
PMID: 19562263
In the majority of the cases cerebrovascular disease is caused by atherosclerosis. Duplexsonography is the diagnostic tool of first choice. Management of cardiovascular risk factors is of paramount importance in...
6.
Gambaryan S, Kobsar A, Hartmann S, Birschmann I, Kuhlencordt P, Muller-Esterl W, et al.
J Thromb Haemost
. 2008 May;
6(8):1376-84.
PMID: 18485089
Objectives: Platelets, specialized adhesive cells, play key roles in normal and pathological hemostasis through their ability to rapidly adhere to subendothelial matrix proteins (adhesion) and to other activated platelets (aggregation),...
7.
Chen J, Kuhlencordt P, Astern J, Gyurko R, Huang P
Circulation
. 2001 Nov;
104(20):2391-4.
PMID: 11705813
Background: Apolipoprotein E (apoE)/endothelial nitric oxide synthase (eNOS) double knockout (DKO) mice demonstrate accelerated atherosclerosis and develop abdominal aortic aneurysms and aortic dissection, suggesting a role for eNOS in suppressing...
8.
Kuhlencordt P, Gyurko R, Han F, Scherrer-Crosbie M, Aretz T, Hajjar R, et al.
Circulation
. 2001 Jul;
104(4):448-54.
PMID: 11468208
Background: To test whether deficiency in endothelial nitric oxide synthase (eNOS) affects atherosclerosis development, we compared lesion formation in apolipoprotein E (apoE)/eNOS-double knockout (DKO) and apoE-knockout (KO) control animals. Methods...
9.
Kuhlencordt P, Chen J, Han F, Astern J, Huang P
Circulation
. 2001 Jun;
103(25):3099-104.
PMID: 11425775
Background: Inducible nitric oxide synthase (iNOS) is expressed by leukocytes and smooth muscle cells in atherosclerotic lesions. To test whether NO produced by iNOS deficiency affects atherosclerosis, we studied apoE/iNOS-double...