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P Hulley

Explore the profile of P Hulley including associated specialties, affiliations and a list of published articles. Areas
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Articles 6
Citations 253
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Recent Articles
1.
Franklin S, Jayadev C, Poulsen R, Hulley P, Price A
Bone Joint Res . 2013 Apr; 1(3):36-40. PMID: 23610669
Objectives: Surgical marking during tendon surgery is often used for technical and teaching purposes. This study investigates the effect of a gentian violet ink marker pen, a common surgical marker,...
2.
Alsousou J, Thompson M, Hulley P, Noble A, Willett K
J Bone Joint Surg Br . 2009 Aug; 91(8):987-96. PMID: 19651823
Although mechanical stabilisation has been a hallmark of orthopaedic surgical management, orthobiologics are now playing an increasing role. Platelet-rich plasma (PRP) is a volume of plasma fraction of autologous blood...
3.
Hulley P, Schachner M, Lubbert H
J Neurosci Res . 1998 Jul; 53(2):129-34. PMID: 9671969
Cell adhesion molecules play a central role in neural development and are also critically involved in axonal regeneration and synaptic plasticity in the adult nervous system. We investigated whether the...
4.
Hulley P, Hartikka J, Abdelal S, Engels P, Buerki H, Wiederhold K, et al.
Eur J Neurosci . 1995 Dec; 7(12):2431-40. PMID: 8845948
The neuropathology of Parkinson's disease is characterized by the degeneration of dopaminergic neurons in the substantia nigra. We have recently shown that the activation of protein kinase A improves the...
5.
Engels P, Abdelal S, Hulley P, Lubbert H
J Neurosci Res . 1995 Jun; 41(2):169-78. PMID: 7650752
We have analyzed the brain distribution of the rat cAMP-specific phosphodiesterases (rPDEIV) which are closely related to the defective gene products of the drosophila melanogaster learning and memory mutant dunce....
6.
Hulley P, Hartikka J, Lubbert H
J Neural Transm Suppl . 1995 Jan; 46:217-28. PMID: 8821058
We have studied how stimulation of protein kinase C and cAMP-dependent protein kinases affect the development of mesencephalic dopaminergic neurons in vitro. IGF-I and bFGF did not activate either second...