P Booms
Overview
Explore the profile of P Booms including associated specialties, affiliations and a list of published articles.
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Articles
22
Citations
527
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0
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Recent Articles
1.
Herrera-Vizcaino C, Dohle E, Al-Maawi S, Booms P, Sader R, Kirkpatrick C, et al.
Eur Cell Mater
. 2019 Apr;
37:250-264.
PMID: 30963526
Different tissue engineering techniques are used to support rapid vascularisation. A novel technique is the use of platelet-rich fibrin (PRF), an autologous source of growth factors. This study was the...
2.
Merritt A, Booms P, Shaw M, Miller D, Daly C, Bilmen J, et al.
Br J Anaesth
. 2017 Apr;
118(4):533-543.
PMID: 28403410
Background: . Missense variants in the ryanodine receptor 1 gene ( RYR1 ) are associated with malignant hyperthermia but only a minority of these have met the criteria for use...
3.
El Bagdadi K, Kubesch A, Yu X, Al-Maawi S, Orlowska A, Dias A, et al.
Eur J Trauma Emerg Surg
. 2017 Mar;
45(3):467-479.
PMID: 28324162
Purpose The present study evaluated the platelet distribution pattern and growth factor release (VEGF, TGF-β1 and EGF) within three PRF (platelet-rich-fibrin) matrices (PRF, A-PRF and A-PRF+) that were prepared using...
4.
Barbeck M, Unger R, Booms P, Dohle E, Sader R, Kirkpatrick C, et al.
J Biomed Mater Res A
. 2016 Jul;
104(12):2928-2935.
PMID: 27419378
The present study analyzes the influence of the addition of monocytes to a biphasic bone substitute with two granule sizes (400-700 μm and 500-1000 μm). The majority of the added...
5.
Barbeck M, Dard M, Kokkinopoulou M, Markl J, Booms P, Sader R, et al.
Biomatter
. 2015 Jun;
5:e1056943.
PMID: 26083163
The present study investigated the influence of granule size of 2 biphasic bone substitutes (BoneCeramic® 400-700 μm and 500-1000 μm) on the induction of multinucleated giant cells (MNGCs) and implant...
6.
Ghanaati S, Barbeck M, Booms P, Lorenz J, Kirkpatrick C, Sader R
Acta Biomater
. 2014 Apr;
10(8):3557-62.
PMID: 24769111
In the present study, the structure of two allogeneic and three xenogeneic bone blocks, which are used in dental and orthopedic surgery, were histologically analyzed. The ultimate goal was to...
7.
Carpenter D, Robinson R, Quinnell R, Ringrose C, Hogg M, Casson F, et al.
Br J Anaesth
. 2009 Aug;
103(4):538-48.
PMID: 19648156
Background: Malignant hyperthermia (MH) is associated, in the majority of cases, with mutations in RYR1, the gene encoding the skeletal muscle ryanodine receptor. Our primary aim was to assess whether...
8.
Robinson R, Carpenter D, Halsall P, Iles D, Booms P, Steele D, et al.
Br J Anaesth
. 2009 May;
103(2):220-5.
PMID: 19454545
Background: Tissue-specific monoallelic silencing of the RYR1 gene has been proposed as an explanation for variable penetrance of dominant RYR1 mutations in malignant hyperthermia (MH). We examined the hypothesis that...
9.
Carpenter D, Morris A, Robinson R, Booms P, Iles D, Halsall P, et al.
Ann Hum Genet
. 2008 Oct;
73(1):10-8.
PMID: 18945287
This study represents a new approach to characterising patients at risk of malignant hyperthermia (MH) through the use of a recently published method for identifying high-risk haplotypes in candidate genes....
10.
Robinson P, Arteaga-Solis E, Baldock C, Collod-Beroud G, Booms P, De Paepe A, et al.
J Med Genet
. 2006 Mar;
43(10):769-87.
PMID: 16571647
Marfan syndrome (MFS), a relatively common autosomal dominant hereditary disorder of connective tissue with prominent manifestations in the skeletal, ocular, and cardiovascular systems, is caused by mutations in the gene...