P A Crossey
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Explore the profile of P A Crossey including associated specialties, affiliations and a list of published articles.
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17
Citations
630
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Recent Articles
1.
Crossey P, Jones J, Miell J
Diabetes
. 2000 Jun;
49(3):457-65.
PMID: 10868969
The insulin/IGF binding protein-1 (IGFBP-1) axis is important in coordinating insulin- and IGF-mediated regulation of glucose metabolism and glycemia. Dysregulation of the axis may play a role in the pathophysiology...
2.
Holt R, Crossey P, Jones J, Baker A, Portmann B, Miell J
Hepatology
. 1997 Dec;
26(6):1600-6.
PMID: 9398004
Major changes in serum levels of insulin-like growth factor I (IGF-I) and IGF-binding proteins (IGFBPs) occur in children with end-stage liver disease in association with changes in body composition. We...
3.
Holt R, Baker A, Jones J, Crossey P, Stone N, Preedy V, et al.
J Endocrinol
. 1996 Jun;
149(3):465-72.
PMID: 8691105
Hepatic gene expression and circulating levels of IGF-binding proteins (IGFBP)-1 to -4 were examined in two rat models of liver disease employing acute hepatitis or chronic structural damage. The study...
4.
Maher E, Webster A, Richards F, Green J, Crossey P, Payne S, et al.
J Med Genet
. 1996 Apr;
33(4):328-32.
PMID: 8730290
Von Hippel-Lindau disease is an autosomal dominantly inherited familial cancer syndrome predisposing to retinal and central nervous system haemangioblastomas, renal cell carcinoma, and phaeochromocytoma. VHL disease shows variable expression and...
5.
Ritter M, Frilling A, Crossey P, Hoppner W, Maher E, Mulligan L, et al.
J Clin Endocrinol Metab
. 1996 Mar;
81(3):1035-7.
PMID: 8772572
Inherited pheochromocytomas are often part of familial syndromes, especially multiple endocrine neoplasia type 2 (MEN 2), retinal cerebellar hemangioblastomatosis [von Hippel-Lindau (vHL) disease] or neurofibromatosis type 1. It is not...
6.
Zbar B, Kishida T, Chen F, Schmidt L, Maher E, Richards F, et al.
Hum Mutat
. 1996 Jan;
8(4):348-57.
PMID: 8956040
Germline mutation analysis was performed in 469 VHL families from North America, Europe, and Japan. Germline mutations were identified in 300/469 (63%) of the families tested; 137 distinct intragenic germline...
7.
Eng C, Crossey P, Mulligan L, Healey C, Houghton C, Prowse A, et al.
J Med Genet
. 1995 Dec;
32(12):934-7.
PMID: 8825918
Phaeochromocytomas may occur sporadically, or as part of the inherited cancer syndromes multiple endocrine neoplasia (MEN) type 2, von Hippel-Lindau disease (VHL), and, rarely, in type 1 neurofibromatosis. In MEN...
8.
Crossey P, Eng C, Ginalska-Malinowska M, Lennard T, Wheeler D, Ponder B, et al.
J Med Genet
. 1995 Nov;
32(11):885-6.
PMID: 8592333
Inherited predisposition to phaeochromocytoma is seen in multiple endocrine neoplasia type 2 syndromes, von Hippel-Lindau (VHL) disease, and neuro-fibromatosis type 1. In addition familial phaeochromocytoma alone has been reported. To...
9.
Neumann H, Eng C, Mulligan L, Glavac D, Zauner I, Ponder B, et al.
JAMA
. 1995 Oct;
274(14):1149-51.
PMID: 7563486
Objective: Multiple endocrine neoplasia, type II (MEN-II) is an autosomal dominant disorder characterized by tumors of thyroid C cells and pheochromocytoma. Recently, germline mutations in the RET proto-oncogene have been...
10.
Foster K, Prowse A, van den Berg A, Fleming S, Hulsbeek M, Crossey P, et al.
Hum Mol Genet
. 1994 Dec;
3(12):2169-73.
PMID: 7881415
Loss of heterozygosity (LOH) studies have suggested that somatic mutations of a tumour suppressor gene or genes on chromosome 3p are a critical event in the pathogenesis of non-familial renal...