Nobuaki Yamanaka
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Explore the profile of Nobuaki Yamanaka including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
532
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Recent Articles
1.
Nagai Y, Yamabe F, Sasaki Y, Ishii T, Nakanishi K, Nakajima K, et al.
Kidney Blood Press Res
. 2020 Jan;
45(2):194-208.
PMID: 31945766
Background: Renin-angiotensin-aldosterone system blockers are known to reduce hypertrophy of vascular smooth muscle cells (SMCs) in hypertensive cases. However, we have reported marked proliferative changes of renal afferent arteriolar SMCs...
2.
Nakanishi K, Nagai Y, Akimoto T, Yamanaka N
J Smooth Muscle Res
. 2017 Mar;
53(0):20-30.
PMID: 28260705
Background Recently, we showed that long-term angiotensin receptor blocker (ARB) administration induced unusual proliferative changes in smooth muscle cells (SMCs) of afferent arterioles of the kidneys of Zucker fatty rats...
3.
Nagai Y, Nakanishi K, Yamanaka N
Kidney Blood Press Res
. 2016 Aug;
41(5):561-569.
PMID: 27553090
Background/aims: We have reported that the long-term administration of angiotensin II receptor blockers (ARBs) induced unusual proliferative changes of renal afferent arteriolar smooth muscle cells (SMCs) in rats, associated with...
4.
Wang P, Kitamura H, Shimizu A, Yamanaka N
Kidney Blood Press Res
. 2015 Apr;
40(2):188-99.
PMID: 25871528
Background/aims: Immunologically and hemodynamically mediated the destruction of glomerular architecture is thought to be the major causes of end-stage renal failure. The purpose of this study is to evaluate the...
5.
Masuda Y, Yamanaka N, Ishikawa A, Kataoka M, Arai T, Wakamatsu K, et al.
Clin Exp Nephrol
. 2014 Jul;
19(3):427-35.
PMID: 25056826
Background: The glomerulus contains well-developed capillaries, which are at risk of injury due to high hydrostatic pressure, hyperfiltration, hypertension and inflammation. However, the pathological alterations of the injured glomerular basement...
6.
Nagai Y, Nakanishi K, Akimoto T, Yamanaka N
J Renin Angiotensin Aldosterone Syst
. 2013 Nov;
15(4):440-9.
PMID: 24222655
Introduction: Our previous study of angiotensin II receptor blocker (ARB) administration in rats induced unusual proliferative changes of smooth muscle cells in renal arteriolar walls. The present study examined if...
7.
Kawano M, Mizushima I, Yamaguchi Y, Imai N, Nakashima H, Nishi S, et al.
Int J Rheumatol
. 2012 Aug;
2012:609795.
PMID: 22899937
Although tubulointerstitial nephritis with IgG4+ plasma cell (PC) infiltration is a hallmark of IgG4-related kidney disease (IgG4-RKD), only a few studies are available about the minimum number of IgG4+ PC...
8.
Kawano M, Saeki T, Nakashima H, Nishi S, Yamaguchi Y, Hisano S, et al.
Nihon Jinzo Gakkai Shi
. 2012 Feb;
53(8):1062-73.
PMID: 22351990
No abstract available.
9.
Kawano M, Saeki T, Nakashima H, Nishi S, Yamaguchi Y, Hisano S, et al.
Clin Exp Nephrol
. 2011 Sep;
15(5):615-626.
PMID: 21898030
Background: IgG4-related disease has attracted wide attention recently. It is characterized by a high level of serum IgG4 and dense infiltration of IgG4-positive plasma cells into multiple organs, with the...
10.
Yamaguchi Y, Kanetsuna Y, Honda K, Yamanaka N, Kawano M, Nagata M
Hum Pathol
. 2011 Sep;
43(4):536-49.
PMID: 21889187
Nephropathy associated with IgG4-related disease is characterized by tubulointerstitial nephritis. To better identify its pathology, the present study analyzed clinicopathologic features of IgG4-related tubulointerstitial nephritis cases from across Japan. Sixteen...