Nellie Georgiou-Karistianis
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Explore the profile of Nellie Georgiou-Karistianis including associated specialties, affiliations and a list of published articles.
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190
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2583
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Recent Articles
1.
Abeyasinghe P, Cole J, Razi A, Poudel G, Paulsen J, Tabrizi S, et al.
Mov Disord
. 2025 Jan;
PMID: 39876588
Background: Despite advancements in understanding Huntington's disease (HD) over the past two decades, absence of disease-modifying treatments remains a challenge. Accurately characterizing progression states is crucial for developing effective therapeutic...
2.
Huynh K, Jamadar S, Lampit A, Siddiqui M, Stout J, Georgiou-Karistianis N
J Neurol
. 2025 Jan;
272(2):159.
PMID: 39847109
Background: Huntington's disease (HD) is a rare neurodegenerative disease that causes progressive cognitive, physical, and psychiatric symptoms. Computerised cognitive training (CCT) is a novel intervention that aims to improve and...
3.
Huynh K, Jamadar S, Stout J, Voigt K, Lampit A, Georgiou-Karistianis N
Neurodegener Dis Manag
. 2025 Jan;
14(6):203-216.
PMID: 39781626
Huntington's disease (HD) causes progressive cognitive decline, with no available treatments. Computerized cognitive training (CCT) has shown efficacy in other populations, but its effects in HD are largely unknown. This...
4.
Ghofrani-Jahromi M, Poudel G, Razi A, Abeyasinghe P, Paulsen J, Tabrizi S, et al.
Neuroimage Clin
. 2024 Aug;
43:103650.
PMID: 39142216
Background: In Huntington's disease clinical trials, recruitment and stratification approaches primarily rely on genetic load, cognitive and motor assessment scores. They focus less on in vivo brain imaging markers, which...
5.
Computerized Cognitive Training Increases Gray Matter Volumes in Huntington's Disease: A Pilot Study
Huynh K, Georgiou-Karistianis N, Lampit A, Siddiqui M, Stout J, Jamadar S
Mov Disord
. 2024 Aug;
39(11):2119-2121.
PMID: 39120126
No abstract available.
6.
Cocozza S, Bosticardo S, Battocchio M, Corben L, Delatycki M, Egan G, et al.
Ann Clin Transl Neurol
. 2024 Jul;
11(7):1691-1702.
PMID: 38952134
Objective: The dentato-thalamo-cortical tract (DTT) is the main cerebellar efferent pathway. Degeneration of the DTT is a core feature of Friedreich ataxia (FRDA). However, it remains unclear whether DTT disruption...
7.
Harding I, Nur Karim M, Selvadurai L, Corben L, Delatycki M, Monti S, et al.
Mov Disord
. 2024 Apr;
39(7):1109-1118.
PMID: 38644761
Background: The dentate nuclei of the cerebellum are key sites of neuropathology in Friedreich ataxia (FRDA). Reduced dentate nucleus volume and increased mean magnetic susceptibility, a proxy of iron concentration,...
8.
The Role of Verbal Fluency in the Cerebellar Cognitive Affective Syndrome Scale in Friedreich Ataxia
Corben L, Blomfield E, Tai G, Bilal H, Harding I, Georgiou-Karistianis N, et al.
Cerebellum
. 2024 Apr;
23(5):1975-1980.
PMID: 38642239
Cerebellar pathology engenders the disturbance of movement that characterizes Friedreich ataxia (FRDA), yet the impact of cerebellar pathology on cognition in FRDA remains unclear. Numerous studies have unequivocally demonstrated the...
9.
Wilkes F, Jakabek D, Walterfang M, Velakoulis D, Poudel G, Stout J, et al.
Psychiatry Res Neuroimaging
. 2023 Sep;
335:111717.
PMID: 37751638
Mapping the spatiotemporal progression of neuroanatomical change in Huntington's Disease (HD) is fundamental to the development of bio-measures for prognostication. Statistical shape analysis to measure the striatum has been performed...
10.
Liu C, Younes L, Tong X, Hinkle J, Wang M, Phatak S, et al.
Brain Commun
. 2023 Sep;
5(5):fcad214.
PMID: 37744022
Huntington's disease is caused by a CAG repeat expansion in the Huntingtin gene (), coding for polyglutamine in the Huntingtin protein, with longer CAG repeats causing earlier age of onset....