N Schreiber-Agus
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Explore the profile of N Schreiber-Agus including associated specialties, affiliations and a list of published articles.
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29
Citations
1547
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Recent Articles
1.
Rose E, Schreiber-Agus N, Bajaj K, Klugman S, Goldwaser T
J Genet Couns
. 2015 Sep;
25(1):18-24.
PMID: 26354339
The Jewish community has traditionally taken ownership of its health, and has taken great strides to raise awareness about genetic issues that affect the community, such as Tay-Sachs disease and...
2.
Barrett M, Shanker V, Severt W, Raymond D, Gross S, Schreiber-Agus N, et al.
JIMD Rep
. 2014 May;
16:31-8.
PMID: 24850235
Mutations in glucosidase, beta, acid (GBA) are associated with cognitive impairment in Parkinson disease (PD) as well as dementia with Lewy bodies. For both of these diseases, dementia and hallucinations...
3.
Barrett M, Hagenah J, Dhawan V, Peng S, Stanley K, Raymond D, et al.
Parkinsonism Relat Disord
. 2012 Oct;
19(2):186-91.
PMID: 23062841
Background: Heterozygous glucocerebrosidase (GBA) mutations are the leading genetic risk factor for Parkinson disease, yet imaging correlates, particularly transcranial sonography, have not been extensively described. Methods: To determine whether GBA...
4.
Pirity M, Blanck J, Schreiber-Agus N
Curr Top Microbiol Immunol
. 2006 Apr;
302:205-34.
PMID: 16620030
The past two decades of gene targeting experiments have allowed us to make significant strides towards understanding how the Myc/Max/Mad network influences multiple aspects of cellular behavior during development. Here...
5.
Park D, Razani B, Lasorella A, Schreiber-Agus N, Pestell R, Iavarone A, et al.
Biochemistry
. 2001 Mar;
40(11):3354-62.
PMID: 11258956
The c-Myc oncoprotein contributes to oncogenesis by activating and repressing a repertoire of genes involved in cellular proliferation, metabolism, and apoptosis. Increasing evidence suggests that the repressor function of c-Myc...
6.
Lu Y, Lian H, Sharma P, Schreiber-Agus N, Russell R, Chin L, et al.
Mol Cell Biol
. 2001 Mar;
21(5):1810-8.
PMID: 11238917
Cells isolated from individuals with Cockayne syndrome (CS) have a defect in transcription-coupled DNA repair, which rapidly corrects certain DNA lesions located on the transcribed strand of active genes. Despite...
7.
Rudolph K, Chang S, Millard M, Schreiber-Agus N, DePinho R
Science
. 2000 Feb;
287(5456):1253-8.
PMID: 10678830
Accelerated telomere loss has been proposed to be a factor leading to end-stage organ failure in chronic diseases of high cellular turnover such as liver cirrhosis. To test this hypothesis...
8.
OHagan R, Schreiber-Agus N, Chen K, David G, Engelman J, Schwab R, et al.
Nat Genet
. 2000 Feb;
24(2):113-9.
PMID: 10655054
Myc and Mad family proteins regulate multiple biological processes through their capacity to influence gene expression directly. Here we show that the basic regions of Myc and Mad proteins are...
9.
Sharma P, Schreiber-Agus N
Oncogene
. 1999 Sep;
18(38):5349-55.
PMID: 10498888
The pathogenetic basis of prostate cancer remains highly elusive; its clarification could be facilitated greatly by laboratory and clinical models of the disease. Although the genetically manipulated mouse has been...
10.
Schreiber-Agus N, DePinho R
Bioessays
. 1998 Nov;
20(10):808-18.
PMID: 9819568
The functions of Myc in transformation and transactivation are countered by the suppressive actions of the Mad(Mxi1) family. Mad(Mxi1) proteins not only compete with Myc for dimerization to Max and...