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N Ben-Chetrit

Explore the profile of N Ben-Chetrit including associated specialties, affiliations and a list of published articles. Areas
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Articles 5
Citations 107
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Recent Articles
1.
Pradeep C, Zeisel A, Kostler W, Lauriola M, Jacob-Hirsch J, Haibe-Kains B, et al.
Oncogene . 2024 Feb; 43(17):1317. PMID: 38418546
No abstract available.
2.
Pareja F, Ferraro D, Rubin C, Cohen-Dvashi H, Zhang F, Aulmann S, et al.
Oncogene . 2011 Dec; 31(43):4599-608. PMID: 22179831
Once stimulated, the epidermal growth factor receptor (EGFR) undergoes self-phosphorylation, which, on the one hand, instigates signaling cascades, and on the other hand, recruits CBL ubiquitin ligases, which mark EGFRs...
3.
Pradeep C, Zeisel A, Kostler W, Lauriola M, Jacob-Hirsch J, Haibe-Kains B, et al.
Oncogene . 2011 Dec; 31(31):3569-83. PMID: 22139081
The HER2/neu oncogene encodes a receptor-like tyrosine kinase whose overexpression in breast cancer predicts poor prognosis and resistance to conventional therapies. However, the mechanisms underlying aggressiveness of HER2 (human epidermal...
4.
Lindzen M, Carvalho S, Starr A, Ben-Chetrit N, Pradeep C, Kostler W, et al.
Oncogene . 2011 Nov; 31(30):3505-15. PMID: 22105361
Epidermal growth factor (EGF)-like growth factors control tumor progression as well as evasion from the toxic effects of chemotherapy. Accordingly, antibodies targeting the cognate receptors, such as EGFR/ErbB-1 and the...
5.
Emde A, Pradeep C, Ferraro D, Ben-Chetrit N, Sela M, Ribba B, et al.
Oncogene . 2010 Dec; 30(14):1631-42. PMID: 21132012
Monoclonal antibodies (mAbs) to HER2 are currently used to treat breast cancer, but low clinical efficacy, along with primary and acquired resistance to therapy, commonly limit clinical applications. We previously...