Mojtaba Zare Ebrahimabad
Overview
Explore the profile of Mojtaba Zare Ebrahimabad including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
4
Citations
14
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Kamil Alhassbalawi N, Zare Ebrahimabad M, Seyedhosseini F, Bagheri Y, Abdollahi N, Nazari A, et al.
Rep Biochem Mol Biol
. 2024 Feb;
12(2):220-232.
PMID: 38317820
Background: Systemic Lupus Erythematosus (SLE) is a chronic autoimmune condition that affects multiple organs significantly impacts morbidity and mortality. The development of SLE is influenced by genetic predisposition and dysregulated...
2.
Alsbihawi T, Zare Ebrahimabad M, Seyedhosseini F, Davoodi H, Abdolahi N, Nazari A, et al.
World J Exp Med
. 2024 Jan;
13(5):102-114.
PMID: 38173550
Background: Systemic lupus erythematosus (SLE) is a chronic autoimmune disorder impacting multiple organs, influenced by genetic factors, especially those related to the immune system. However, there is a need for...
3.
Jazi M, Mohammadi S, Zare Ebrahimabad M, Sedighi S, Abdolahi N, Tabarraei A, et al.
J Biochem Mol Toxicol
. 2021 Sep;
35(12):e22916.
PMID: 34580959
Background: Genetic variations of aryl hydrocarbon receptor (AHR) pathway genes could influence the imbalanced immune response to xenobiotics. Therefore, we aimed to investigate the polymorphism of AHR pathway genes in...
4.
Mohammadi S, Jazi M, Zare Ebrahimabad M, Eghbalpour F, Abdolahi N, Tabarraei A, et al.
Int J Immunogenet
. 2018 Nov;
46(1):20-30.
PMID: 30430731
Systemic lupus erythematosus (SLE) is a chronic autoimmune disease with unknown aetiology. According to the role of interleukin 10 (IL10) in SLE pathogenesis, the genetic alterations in its promoter region...