Mochtar Pribadi
Overview
Explore the profile of Mochtar Pribadi including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
671
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Recent Articles
1.
Abujarour R, Dinella J, Pribadi M, Fong L, Denholtz M, Gutierrez A, et al.
Future Sci OA
. 2024 May;
10(1):FSO964.
PMID: 38817352
We explored the generation of human induced pluripotent stem cells (iPSCs) solely through the transcriptional activation of endogenous genes by CRISPR activation (CRISPRa). Minimal number of human-specific guide RNAs targeting...
2.
Goulding J, Yeh W, Hancock B, Blum R, Xu T, Yang B, et al.
Med
. 2023 May;
4(7):457-477.e8.
PMID: 37172578
Background: The advent of chimeric antigen receptor (CAR) T cell therapies has transformed the treatment of hematological malignancies; however, broader therapeutic success of CAR T cells has been limited in...
3.
Huang A, Taylor A, Ghogha A, Pribadi M, Wang Q, Kim T, et al.
J Neurosci Res
. 2020 Aug;
100(1):19-34.
PMID: 32830380
The actions of endogenous opioids and nociceptin/orphanin FQ are mediated by four homologous G protein-coupled receptors that constitute the opioid receptor family. However, little is known about opioid systems in...
4.
Steele N, Bright A, Lee S, Fong J, Bonham L, Karydas A, et al.
Adv Genomics Genet
. 2019 Apr;
8:23-33.
PMID: 31031559
Background: Frontotemporal lobar degeneration (FTLD) is a leading cause of dementia, and elucidating its genetic underpinnings is critical. FTLD research centers typically recruit patient cohorts that are limited by the...
5.
Lopez-Gonzalez R, Yang D, Pribadi M, Kim T, Krishnan G, Choi S, et al.
Proc Natl Acad Sci U S A
. 2019 Apr;
116(19):9628-9633.
PMID: 31019093
GGGGCC (GC) repeat expansion in is the most common genetic cause of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). One class of major pathogenic molecules in -ALS/FTD is dipeptide...
6.
Lee S, Sias A, Mandelli M, Brown J, Brown A, Khazenzon A, et al.
Neuroimage Clin
. 2017 Mar;
14:286-297.
PMID: 28337409
Hexanucleotide repeat expansions in are the most common known genetic cause of familial and sporadic frontotemporal dementia and amyotrophic lateral sclerosis. Previous work has shown that patients with behavioral variant...
7.
Wang X, Hao L, Saur T, Joyal K, Zhao Y, Zhai D, et al.
Front Mol Neurosci
. 2016 Nov;
9:113.
PMID: 27877110
An abnormally expanded GGGGCC repeat in C9ORF72 is the most frequent causal mutation associated with amyotrophic lateral sclerosis (ALS)/frontotemporal lobar degeneration (FTLD). Both () and () mechanisms have been involved...
8.
Vatsavayai S, Yoon S, Gardner R, Gendron T, Vargas J, Trujillo A, et al.
Brain
. 2016 Nov;
139(Pt 12):3202-3216.
PMID: 27797809
SEE SCABER AND TALBOT DOI101093/AWW264 FOR A SCIENTIFIC COMMENTARY ON THIS ARTICLE: A GGGGCC repeat expansion in C9orf72 leads to frontotemporal dementia and/or amyotrophic lateral sclerosis. Diverse pathological features have...
9.
Swartz E, Baek J, Pribadi M, Wojta K, Almeida S, Karydas A, et al.
Stem Cells Transl Med
. 2016 Jul;
5(11):1461-1472.
PMID: 27369896
Significance: Protocols to produce skeletal myotubes for disease modeling or therapy are scarce and incomplete. The present study efficiently generates functional skeletal myotubes from human induced pluripotent stem cells using...
10.
Watson A, Pribadi M, Chowdari K, Clifton S, Wood J, Miller B, et al.
Psychiatry Res
. 2016 Jan;
235:200-2.
PMID: 26723138
A pathologic hexanucleotide repeat expansion in C9orf72 causes frontotemporal dementia (FTD) or amyotrophic lateral sclerosis (ALS). Behavioral abnormalities can also occur among mutation carriers with FTD, but it is uncertain...