Mirjam B Smeets
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Explore the profile of Mirjam B Smeets including associated specialties, affiliations and a list of published articles.
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21
Citations
1238
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Recent Articles
1.
Ellenbroek G, de Haan J, van Klarenbosch B, Brans M, van de Weg S, Smeets M, et al.
Sci Rep
. 2017 Dec;
7(1):18039.
PMID: 29269840
Heart failure after myocardial infarction (MI) depends on infarct size and adverse left ventricular (LV) remodelling, both influenced by the inflammatory response. Leukocyte-associated immunoglobulin-like receptor 1 (LAIR-1) is an inhibitory...
2.
Leenders G, Smeets M, van den Boomen M, Berben M, Nabben M, van Strijp D, et al.
Arterioscler Thromb Vasc Biol
. 2017 Nov;
38(1):186-194.
PMID: 29146749
Objective: The endothelium has a crucial role in wound healing, acting as a barrier to control transit of leukocytes. Endothelial barrier function is impaired in atherosclerosis preceding myocardial infarction (MI)....
3.
Koole D, van Herwaarden J, Schalkwijk C, Lafeber F, Vink A, Smeets M, et al.
J Vasc Surg
. 2016 Sep;
65(5):1493-1503.e3.
PMID: 27624703
Background: Diabetes is a risk factor for atherosclerotic disease but negatively associated with the development and progression of abdominal aortic aneurysm (AAA). Advanced glycation end products (AGEs) are increased in...
4.
Buljubasic N, Oemrawsingh R, Smeets M, Cheng J, Regar E, van Geuns R, et al.
Int J Cardiol
. 2016 Jul;
221:682-7.
PMID: 27423090
Background: Conflicting results exist regarding the association between a common Haptoglobin (Hp) polymorphism and risk of coronary artery disease. We investigated the association of three functionally different anti-oxidant and anti-inflammatory...
5.
de Hoog V, Timmers L, van Duijvenvoorde A, de Jager S, van Middelaar B, Smeets M, et al.
Cardiovasc Res
. 2014 Jun;
103(4):521-9.
PMID: 24935433
Aims: Early reperfusion is mandatory for the treatment of acute myocardial infarction. This process, however, also induces additional loss of viable myocardium, called ischaemia-reperfusion (IR) injury. Complement activation plays an...
6.
Arslan F, Lai R, Smeets M, Akeroyd L, Choo A, Aguor E, et al.
Stem Cell Res
. 2013 Feb;
10(3):301-12.
PMID: 23399448
We have previously identified exosomes as the paracrine factor secreted by mesenchymal stem cells. Recently, we found that the key features of reperfusion injury, namely loss of ATP/NADH, increased oxidative...
7.
Arslan F, Smeets M, Buttari B, Profumo E, Rigano R, Akeroyd L, et al.
J Mol Cell Cardiol
. 2013 Jan;
56:116-28.
PMID: 23274064
Decreased haptoglobin (Hp) functionality due to allelic variations is associated with worsened outcome in patients after myocardial infarction (MI). However, mechanisms through which haptoglobin deficiency impairs cardiac repair remain to...
8.
Arslan F, Houtgraaf J, Keogh B, Kazemi K, De Jong R, McCormack W, et al.
Circ Cardiovasc Interv
. 2012 Feb;
5(2):279-87.
PMID: 22354933
Background: Toll-like receptor (TLR)-2 is an important mediator of innate immunity and ischemia/reperfusion-induced cardiac injury. We have previously shown that TLR2 inhibition reduces infarct size and improves cardiac function in...
9.
Arslan F, Smeets M, Riem Vis P, Karper J, Quax P, Bongartz L, et al.
Circ Res
. 2011 Feb;
108(5):582-92.
PMID: 21350212
Rationale: The extracellular matrix may induce detrimental inflammatory responses on degradation, causing adverse cardiac remodeling and heart failure. The extracellular matrix protein fibronectin-EDA (EIIIA; EDA) is upregulated after tissue injury...
10.
Arslan F, Smeets M, ONeill L, Keogh B, McGuirk P, Timmers L, et al.
Circulation
. 2009 Dec;
121(1):80-90.
PMID: 20026776
Background: Reperfusion therapy for myocardial infarction is hampered by detrimental inflammatory responses partly via Toll-like receptor (TLR) activation. Targeting TLR signaling may optimize reperfusion therapy and enhance cell survival and...