Michito Hirakata
Overview
Explore the profile of Michito Hirakata including associated specialties, affiliations and a list of published articles.
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Articles
41
Citations
652
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0
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Recent Articles
1.
Takada K, Katada Y, Ito S, Hayashi T, Kishi J, Itoh K, et al.
Rheumatology (Oxford)
. 2019 Sep;
59(5):1084-1093.
PMID: 31539061
Objective: Interstitial pneumonia is common and has high short-term mortality in patients with PM and DM despite glucocorticoid (GC) treatment. Retrospective studies suggested that the early use of immunosuppressive drugs...
2.
Takada T, Hirakata M, Suwa A, Kaneko Y, Kuwana M, Ishihara T, et al.
Mod Rheumatol
. 2017 Sep;
19(2):165.
PMID: 28925310
No abstract available.
3.
Hanaoka H, Kaneko Y, Suzuki S, Takada T, Hirakata M, Takeuchi T, et al.
Mod Rheumatol
. 2015 Dec;
26(6):979-980.
PMID: 26707188
No abstract available.
4.
Kaneko Y, Hanaoka H, Hirakata M, Takeuchi T, Kuwana M
Rheumatology (Oxford)
. 2014 Feb;
53(6):1120-4.
PMID: 24501248
Objective: The aim of this study was to characterize arthropathies of the hands associated with antiaminoacyl tRNA synthetase (ARS) autoantibodies. Methods: Fifty-six patients with anti-ARS antibodies were selected from consecutive...
5.
Nakashima R, Imura Y, Hosono Y, Seto M, Murakami A, Watanabe K, et al.
PLoS One
. 2014 Jan;
9(1):e85062.
PMID: 24454792
Objective: Autoantibodies to aminoacyl-tRNA synthetases (ARSs) are useful in the diagnosis of idiopathic inflammatory myopathy (IIM) with interstitial pneumonia (IP). We developed an enzyme-linked immunosorbent assay (ELISA) system using a...
6.
Shimane K, Kochi Y, Suzuki A, Okada Y, Ishii T, Horita T, et al.
Rheumatology (Oxford)
. 2013 Feb;
52(7):1172-82.
PMID: 23407388
Objective. To re-evaluate the roles of HLA-DRB1 alleles in susceptibility to SLE and RA and their effects on autoantibody status in large-scale Japanese cohorts. Methods. A total of 656 SLE,...
7.
Okada Y, Shimane K, Kochi Y, Tahira T, Suzuki A, Higasa K, et al.
PLoS Genet
. 2012 Feb;
8(1):e1002455.
PMID: 22291604
Systemic lupus erythematosus (SLE) is an autoimmune disease that causes multiple organ damage. Although recent genome-wide association studies (GWAS) have contributed to discovery of SLE susceptibility genes, few studies has...
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