Michael M Kaminski
Overview
Explore the profile of Michael M Kaminski including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
19
Citations
584
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
11.
Kaminski M, Alcantar M, Lape I, Greensmith R, Huske A, Valeri J, et al.
Nat Biomed Eng
. 2020 Apr;
4(6):601-609.
PMID: 32284553
In organ transplantation, infection and rejection are major causes of graft loss. They are linked by the net state of immunosuppression. To diagnose and treat these conditions earlier, and to...
12.
Lagies S, Pichler R, Bork T, Kaminski M, Troendle K, Zimmermann S, et al.
Cells
. 2019 Sep;
8(10).
PMID: 31554337
Diabetic kidney disease is a major complication in diabetes mellitus, and the most common reason for end-stage renal disease. Patients suffering from diabetes mellitus encounter glomerular damage by basement membrane...
13.
Jansen K, Castilho M, Aarts S, Kaminski M, Lienkamp S, Pichler R, et al.
Macromol Biosci
. 2018 Dec;
19(2):e1800412.
PMID: 30548802
The increasing prevalence of end-stage renal disease and persistent shortage of donor organs call for alternative therapies for kidney patients. Dialysis remains an inferior treatment as clearance of large and...
14.
Lagies S, Pichler R, Kaminski M, Schlimpert M, Walz G, Lienkamp S, et al.
Sci Rep
. 2018 Mar;
8(1):3878.
PMID: 29497074
Fibroblasts can be directly reprogrammed to induced renal tubular epithelial cells (iRECs) using four transcription factors. These engineered cells may be used for disease modeling, cell replacement therapy or drug...
15.
Kaminski M, Tosic J, Pichler R, Arnold S, Lienkamp S
Cell Tissue Res
. 2017 Jun;
369(1):185-197.
PMID: 28560692
Growing knowledge of how cell identity is determined at the molecular level has enabled the generation of diverse tissue types, including renal cells from pluripotent or somatic cells. Recently, several...
16.
Vivante A, Mann N, Yonath H, Weiss A, Getwan M, Kaminski M, et al.
J Am Soc Nephrol
. 2017 Apr;
28(8):2364-2376.
PMID: 28381549
Congenital anomalies of the kidney and urinary tract (CAKUT) are the most common cause of CKD in the first three decades of life. However, for most patients with CAKUT, the...
17.
Kaminski M, Tosic J, Kresbach C, Engel H, Klockenbusch J, Muller A, et al.
Nat Cell Biol
. 2016 Nov;
18(12):1269-1280.
PMID: 27820600
Direct reprogramming by forced expression of transcription factors can convert one cell type into another. Thus, desired cell types can be generated bypassing pluripotency. However, direct reprogramming towards renal cells...
18.
Vivante A, Kleppa M, Schulz J, Kohl S, Sharma A, Chen J, et al.
Am J Hum Genet
. 2015 Aug;
97(2):291-301.
PMID: 26235987
Congenital anomalies of the kidneys and urinary tract (CAKUT) are the most common cause of chronic kidney disease in the first three decades of life. Identification of single-gene mutations that...
19.
Kaminski M, Ohnemus A, Staeheli P, Rubbenstroth D
J Virol
. 2012 Nov;
87(3):1912-5.
PMID: 23192869
Resistance of influenza A viruses to neuraminidase inhibitors can arise through mutations in the neuraminidase (NA) gene. We show here that a Q136K mutation in the NA of the 2009...
20.
Kaminski M, Ohnemus A, Cornitescu M, Staeheli P
J Gen Virol
. 2011 Dec;
93(Pt 3):555-559.
PMID: 22170637
Types I and III interferons (IFNs) elicit protective antiviral immune responses during influenza virus infection. Although many cell types can synthesize IFN in response to virus infection, it remains unclear...