Michael D Stutz
Overview
Explore the profile of Michael D Stutz including associated specialties, affiliations and a list of published articles.
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20
Citations
991
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Recent Articles
1.
Pidot S, Klatt S, Ates L, Frigui W, Sayes F, Majlessi L, et al.
PLoS Pathog
. 2024 Aug;
20(8):e1012440.
PMID: 39207937
Reconstructing the evolutionary origins of Mycobacterium tuberculosis, the causative agent of human tuberculosis, has helped identify bacterial factors that have led to the tubercle bacillus becoming such a formidable human...
2.
Preston S, Stutz M, Allison C, Nachbur U, Gouil Q, Tran B, et al.
Gastroenterology
. 2022 Aug;
163(6):1643-1657.e14.
PMID: 36037995
Background & Aims: Necroptosis is a highly inflammatory mode of cell death that has been implicated in causing hepatic injury including steatohepatitis/ nonalcoholic steatohepatitis (NASH); however, the evidence supporting these...
3.
M Bader S, Preston S, Saliba K, Lipszyc A, Grant Z, Mackiewicz L, et al.
Cell Death Differ
. 2022 Jul;
30(1):27-36.
PMID: 35871233
Caspase-8 transduces signals from death receptor ligands, such as tumor necrosis factor, to drive potent responses including inflammation, cell proliferation or cell death. This is a developmentally essential function because...
4.
Ebrahimnezhaddarzi S, Bird C, Allison C, Tuipulotu D, Kostoulias X, Macri C, et al.
Immunol Cell Biol
. 2022 Apr;
100(7):529-546.
PMID: 35471730
To control infections phagocytes can directly kill invading microbes. Macrophage-expressed gene 1 (Mpeg1), a pore-forming protein sometimes known as perforin-2, is reported to be essential for bacterial killing following phagocytosis....
5.
Teh C, Preston S, Robbins A, Stutz M, Cooney J, Clark M, et al.
Sci Immunol
. 2022 Mar;
7(69):eabn8041.
PMID: 35333545
Targeting the potent immunosuppressive properties of FOXP3 regulatory T cells (T) has substantial therapeutic potential for treating autoimmune and inflammatory diseases. Yet, the molecular mechanisms controlling T homeostasis, particularly during...
6.
Clark M, Huynh T, Rao S, Mackiewicz L, Mason H, Romal S, et al.
Cell Death Dis
. 2021 Jun;
12(7):641.
PMID: 34162831
A major unmet clinical need is a therapeutic capable of removing hepatitis B virus (HBV) genome from the liver of infected individuals to reduce their risk of developing liver cancer....
7.
Hildebrand J, Kauppi M, Majewski I, Liu Z, Cox A, Miyake S, et al.
Nat Commun
. 2020 Jun;
11(1):3150.
PMID: 32561755
MLKL is the essential effector of necroptosis, a form of programmed lytic cell death. We have isolated a mouse strain with a single missense mutation, Mlkl, that alters the two-helix...
8.
Stutz M, Pellegrini M
Trends Microbiol
. 2018 Aug;
26(10):813-814.
PMID: 30119946
Mycobacterium tuberculosis interferes with the ability of its host cell to undergo apoptosis. Arnett et al. report that the pathogen promotes macrophage survival by engaging the nuclear receptor PPARγ to...
9.
Seillet C, Arvell E, Lacey D, Stutz M, Pellegrini M, Whitehead L, et al.
Immunol Cell Biol
. 2018 Aug;
97(1):29-38.
PMID: 30107066
BPSM1 (Bone phenotype spontaneous mutant 1) mice develop severe polyarthritis and heart valve disease as a result of a spontaneous mutation in the Tnf gene. In these mice, the insertion...
10.
Stutz M, Ojaimi S, Ebert G, Pellegrini M
Front Immunol
. 2018 Jun;
9:1178.
PMID: 29892302
The dwindling list of antimicrobial agents exhibiting broad efficacy against clinical strains of (Mtb) has forced the medical community to redefine current approaches to the treatment of tuberculosis (TB). Host...