Melanie Ehrlich
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Explore the profile of Melanie Ehrlich including associated specialties, affiliations and a list of published articles.
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3226
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Recent Articles
1.
Ehrlich M, Ehrlich K, Lacey M, Baribault C, Sen S, Esteve P, et al.
Epigenomes
. 2024 Feb;
8(1).
PMID: 38390894
While studying myoblast methylomes and transcriptomes, we found that had a remarkable preference for expression in both myoblasts and cerebellum. To understand how widespread such a relationship was and its...
2.
Ehrlich K, Lacey M, Baribault C, Sen S, Esteve P, Pradhan S, et al.
Epigenomes
. 2022 Dec;
6(4).
PMID: 36547252
TBX15, which encodes a differentiation-related transcription factor, displays promoter-adjacent DNA hypermethylation in myoblasts and skeletal muscle (psoas) that is absent from non-expressing cells in other lineages. By whole-genome bisulfite sequencing...
3.
Ehrlich M
Epigenomics
. 2022 Mar;
14(6):351-358.
PMID: 35255735
Melanie Ehrlich, PhD, is a professor in the Tulane Cancer Center, the Tulane Center for Medical Bioinformatics and Genomics and the Hayward Human Genetics Program at Tulane Medical School, New...
4.
Zhang X, Li T, Xiao H, Ehrlich K, Shen H, Deng H, et al.
Int J Mol Sci
. 2022 Feb;
23(3).
PMID: 35163195
Concern about rising rates of obesity has prompted searches for obesity-related single nucleotide polymorphisms (SNPs) in genome-wide association studies (GWAS). Identifying plausible regulatory SNPs is very difficult partially because of...
5.
Ehrlich K, Deng H, Ehrlich M
Epigenomes
. 2022 Jan;
6(1).
PMID: 35076500
Striated muscle has especially large energy demands. We identified 97 genes preferentially expressed in skeletal muscle and heart, but not in aorta, and found significant enrichment for mitochondrial associations among...
6.
Ehrlich K, Lacey M, Ehrlich M
Epigenomes
. 2021 Dec;
4(1).
PMID: 34968235
Much remains to be discovered about the intersection of tissue-specific transcription control and the epigenetics of skeletal muscle (SkM), a very complex and dynamic organ. From four gene families, Leucine-Rich...
7.
Zhang X, Deng H, Shen H, Ehrlich M
JBMR Plus
. 2021 May;
5(5):e10481.
PMID: 33977200
Genetic risk factors for osteoporosis, a prevalent disease associated with aging, have been examined in many genome-wide association studies (GWASs). A major challenge is to prioritize transcription-regulatory GWAS-derived variants that...
8.
Chandra S, Ehrlich K, Lacey M, Baribault C, Ehrlich M
Epigenomics
. 2021 Feb;
13(3):219-234.
PMID: 33538177
Excessive inflammatory signaling and pathological remodeling of the extracellular matrix drive cardiac fibrosis and require changes in gene expression. Using bioinformatics, both tissue-specific expression profiles and epigenomic profiles of some...
9.
Ehrlich K, Baribault C, Ehrlich M
Int J Mol Sci
. 2020 Nov;
21(21).
PMID: 33182325
and the related genes encode components of the Cullin-E3 ubiquitin ligase complex and typically target tissue-specific proteins for degradation, thereby affecting differentiation, homeostasis, metabolism, cell signaling, and the oxidative stress...
10.
Zhang X, Ehrlich K, Yu F, Hu X, Meng X, Deng H, et al.
Epigenetics
. 2020 Jan;
15(6-7):728-749.
PMID: 31975641
A major challenge in translating findings from genome-wide association studies (GWAS) to biological mechanisms is pinpointing functional variants because only a very small percentage of variants associated with a given...