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Meghan C Campbell

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Articles 48
Citations 1081
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Recent Articles
1.
Baudendistel S, Rawson K, Lessov-Schlaggar C, Maiti B, Kotzbauer P, Perlmutter J, et al.
Clin Biomech (Bristol) . 2025 Feb; 122:106445. PMID: 39903964
Background: Clinical subtypes in Parkinson's disease including non-motor manifestations may be more beneficial than subtypes based upon motor manifestations alone. Inclusion of gait metrics may help identity targets for rehabilitation...
2.
Cash T, Lessov-Schlaggar C, Foster E, Myers P, Jackson J, Maiti B, et al.
Parkinsonism Relat Disord . 2024 Jun; 124:107016. PMID: 38838453
Background: We recently identified three distinct Parkinson's disease subtypes: "motor only" (predominant motor deficits with intact cognition and psychiatric function); "psychiatric & motor" (prominent psychiatric symptoms and moderate motor deficits);...
3.
ODonnell J, Soda A, Jiang H, Norris S, Maiti B, Karimi M, et al.
J Nucl Med . 2024 Apr; 65(6):956-961. PMID: 38604762
Molecular imaging of brain vesicular acetylcholine transporter provides a biomarker to explore cholinergic systems in humans. We aimed to characterize the distribution of, and optimize methods to quantify, the vesicular...
4.
Liu G, Ni C, Zhan J, Li W, Luo J, Liao Z, et al.
Brain . 2022 Nov; 146(1):42-49. PMID: 36343661
Mitochondria are a culprit in the onset of Parkinson's disease, but their role during disease progression is unclear. Here we used Cox proportional hazards models to exam the effect of...
5.
Martin W, Younce J, Campbell M, Racette B, Norris S, Ushe M, et al.
Ann Neurol . 2022 Nov; 93(1):184-195. PMID: 36331161
Objective: The objective of this study was to evaluate the relationship between Parkinson's disease (PD) with dementia and cortical proteinopathies in a large population of pathologically confirmed patients with PD....
6.
Myers P, ODonnell J, Jackson J, Lessov-Schlaggar C, Miller R, Foster E, et al.
Neurology . 2022 Apr; 99(1):e66-e76. PMID: 35418463
Background And Objectives: People with Parkinson disease (PD) commonly experience cognitive decline, which may relate to increased α-synuclein, tau, and β-amyloid accumulation. This study examines whether the different proteins predict...
7.
Miller R, Dhavale D, OShea J, Andruska K, Liu J, Franklin E, et al.
Ann Clin Transl Neurol . 2022 Jan; 9(2):106-121. PMID: 35060360
Objective: Parkinson disease (PD) is defined by the accumulation of misfolded α-synuclein (α-syn) in Lewy bodies and Lewy neurites. It affects multiple cortical and subcortical neuronal populations. The majority of...
8.
Myers P, Jackson J, Clover A, Lessov-Schlaggar C, Foster E, Maiti B, et al.
Ann Clin Transl Neurol . 2021 Jul; 8(8):1695-1708. PMID: 34310084
Objective: To examine specific symptom progression patterns and possible disease staging in Parkinson disease clinical subtypes. Methods: We recently identified Parkinson disease clinical subtypes based on comprehensive behavioral evaluations, "Motor...
9.
Maiti B, Rawson K, Tanenbaum A, Koller J, Snyder A, Campbell M, et al.
Mov Disord . 2021 Jun; 36(11):2559-2568. PMID: 34109682
Background: Dysfunction of cerebellar vermis contributes to gait abnormalities in multiple conditions and may play a key role in gait impairment in Parkinson's disease (PD). Objective: The purpose of this...
10.
Horin A, Myers P, Pickett K, Earhart G, Campbell M
Behav Brain Res . 2021 Jun; 411:113398. PMID: 34087255
Introduction: Parkinson's disease (PD) is a movement disorder caused by dysfunction in the basal ganglia (BG). Clinically relevant gait deficits, such as decreased velocity and increased variability, may be caused...