Matthias Dewenter
Overview
Explore the profile of Matthias Dewenter including associated specialties, affiliations and a list of published articles.
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Articles
31
Citations
655
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Recent Articles
1.
Gjerga E, Dewenter M, Britto-Borges T, Grosso J, Stein F, Eschenbach J, et al.
Database (Oxford)
. 2024 Jul;
2024.
PMID: 39051433
Time-course multi-omics data of a murine model of progressive heart failure (HF) induced by transverse aortic constriction (TAC) provide insights into the molecular mechanisms that are causatively involved in contractile...
2.
Dewenter M, Seitz T, Steinbrecher J, Westenbrink B, Ling H, Lehnart S, et al.
ESC Heart Fail
. 2024 Apr;
11(4):2191-2199.
PMID: 38616546
Aims: Hyperactivity of Ca/calmodulin-dependent protein kinase II (CaMKII) has emerged as a central cause of pathologic remodelling in heart failure. It has been suggested that CaMKII-induced hyperphosphorylation of the ryanodine...
3.
Stellato M, Dewenter M, Rudnik M, Hukara A, Ozsoy C, Renoux F, et al.
Commun Biol
. 2023 Feb;
6(1):213.
PMID: 36823451
No abstract available.
4.
Stellato M, Dewenter M, Rudnik M, Hukara A, Ozsoy C, Renoux F, et al.
Commun Biol
. 2023 Feb;
6(1):161.
PMID: 36759717
Fibrotic changes in the myocardium and cardiac arrhythmias represent fatal complications in systemic sclerosis (SSc), however the underlying mechanisms remain elusive. Mice overexpressing transcription factor Fosl-2 (Fosl-2) represent animal model...
5.
Cachorro E, Gunscht M, Schubert M, Sadek M, Siegert J, Dutt F, et al.
Circ Res
. 2023 Jan;
132(4):400-414.
PMID: 36715019
Background: Ventricular arrhythmia and sudden cardiac death are the most common lethal complications after myocardial infarction. Antiarrhythmic pharmacotherapy remains a clinical challenge and novel concepts are highly desired. Here, we...
6.
Lopez M, Malacarne P, Ramanujam D, Warwick T, Muller N, Hu J, et al.
Front Physiol
. 2022 Dec;
13:1056369.
PMID: 36531184
The cytochrome P450 reductase (POR) transfers electrons to all microsomal cytochrome P450 enzymes (CYP450) thereby driving their activity. In the vascular system, the POR/CYP450 system has been linked to the...
7.
Lennermann D, Pepin M, Grosch M, Konrad L, Kemmling E, Hartmann J, et al.
Am J Physiol Heart Circ Physiol
. 2022 Nov;
323(6):H1296-H1310.
PMID: 36367695
cardiomyopathy is an arrhythmogenic form of dilated cardiomyopathy caused by mutations in the splicing factor RBM20. A recent study found a more severe phenotype in male patients with cardiomyopathy patients...
8.
Bruns B, Daub R, Schmitz T, Hamze-Sinno M, Spaich S, Dewenter M, et al.
Basic Res Cardiol
. 2022 Sep;
117(1):44.
PMID: 36068417
Myocardial infarction (MI) with subsequent depression is associated with increased cardiac mortality. Impaired central mineralocorticoid (MR) and glucocorticoid receptor (GR) equilibrium has been suggested as a key mechanism in the...
9.
Dewenter M, Pan J, Knodler L, Tzschockel N, Henrich J, Cordero J, et al.
Basic Res Cardiol
. 2022 Mar;
117(1):15.
PMID: 35286475
Hyperactivity of the sympathetic nervous system is a major driver of cardiac remodeling, exerting its effects through both α-, and β-adrenoceptors (α-, β-ARs). As the relative contribution of subtype α-AR...
10.
Weis E, Puchalska P, Nelson A, Taylor J, Moll I, Hasan S, et al.
EMBO Mol Med
. 2022 Feb;
14(4):e14753.
PMID: 35179309
Blood vessel formation is dependent on metabolic adaption in endothelial cells. Glucose and fatty acids are essential substrates for ATP and biomass production; however, the metabolism of other substrates remains...