Masayoshi Kamon
Overview
Explore the profile of Masayoshi Kamon including associated specialties, affiliations and a list of published articles.
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Articles
11
Citations
115
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0
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Recent Articles
1.
Kamon M, Wakatsuki S, Nakamori M, Takahashi M, Mori-Yoshimura M, Komaki H, et al.
Hum Mol Genet
. 2024 Dec;
34(4):327-337.
PMID: 39679849
Myotonic dystrophy type 1 (DM1) is a dominantly inherited multi-system disease caused by expanded CTG repeats in the 3' untranslated region of the dystrophia myotonica protein kinase (DMPK) gene. Similar...
2.
Takezawa Y, Kamon M, Hiraki-Kamon K, Mitani A, Shiraishi A, Kato H
Exp Eye Res
. 2024 Apr;
243:109916.
PMID: 38679224
The conjunctiva is a non-keratinized, stratified columnar epithelium with characteristics different from the cornea and eyelid epidermis. From development to adulthood, a distinguishing feature of ocular versus epidermal epithelia is...
3.
Zhang S, Ohkawara B, Ito M, Huang Z, Zhao F, Nakata T, et al.
Hum Mol Genet
. 2022 Dec;
32(9):1511-1523.
PMID: 36579833
At the neuromuscular junction, the downstream of tyrosine kinase 7 (DOK7) enhances the phosphorylation of muscle-specific kinase (MuSK) and induces clustering of acetylcholine receptors (AChRs). We identified a patient with...
4.
Huang K, Masuda A, Chen G, Bushra S, Kamon M, Araki T, et al.
Sci Rep
. 2020 Feb;
10(1):2558.
PMID: 32054946
Muscleblind-like 1 (MBNL1) is a ubiquitously expressed RNA-binding protein, which is highly expressed in skeletal muscle. Abnormally expanded CUG-repeats in the DMPK gene cause myotonic dystrophy type 1 (DM1) by...
5.
Ueki J, Nakamori M, Nakamura M, Nishikawa M, Yoshida Y, Tanaka A, et al.
Sci Rep
. 2017 Feb;
7:42522.
PMID: 28211918
Myotonic dystrophy type 1 (DM1) is an autosomal-dominant multi-system disease caused by expanded CTG repeats in dystrophia myotonica protein kinase (DMPK). The expanded CTG repeats are unstable and can increase...
6.
Hirasaki M, Hiraki-Kamon K, Kamon M, Suzuki A, Katano M, Nishimoto M, et al.
PLoS One
. 2014 Jan;
8(12):e83769.
PMID: 24386274
Predominant transcriptional subnetworks called Core, Myc, and PRC modules have been shown to participate in preservation of the pluripotency and self-renewality of embryonic stem cells (ESCs). Epiblast stem cells (EpiSCs)...
7.
Kamon M, Katano M, Hiraki-Kamon K, Hishida T, Nakachi Y, Mizuno Y, et al.
Stem Cells Dev
. 2013 Nov;
23(18):2170-9.
PMID: 24200330
Somatic cells can be reprogrammed to induced pluripotent stem cells (iPSCs) by defined factors. However, substantial cell numbers subjected to iPSC induction stray from the main reprogramming route and are...
8.
Nishimoto M, Katano M, Yamagishi T, Hishida T, Kamon M, Suzuki A, et al.
PLoS One
. 2013 Jul;
8(7):e68119.
PMID: 23874519
Embryogenesis in placental mammals is sustained by exquisite interplay between the embryo proper and placenta. UTF1 is a developmentally regulated gene expressed in both cell lineages. Here, we analyzed the...
9.
Hikichi T, Matoba R, Ikeda T, Watanabe A, Yamamoto T, Yoshitake S, et al.
Proc Natl Acad Sci U S A
. 2013 Apr;
110(16):6412-7.
PMID: 23550161
Transcription factors (TFs) are able to regulate differentiation-related processes, including dedifferentiation and direct conversion, through the regulation of cell type-specific transcriptional profiles. However, the functional interactions between the TFs regulating...
10.
Hishida T, Nozaki Y, Nakachi Y, Mizuno Y, Iseki H, Katano M, et al.
Stem Cells
. 2012 Jun;
30(8):1634-44.
PMID: 22696478
c-Myc participates in diverse cellular processes including cell cycle control, tumorigenic transformation, and reprogramming of somatic cells to induced pluripotent cells. c-Myc is also an important regulator of self-renewal and...