Masanori Adachi
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Explore the profile of Masanori Adachi including associated specialties, affiliations and a list of published articles.
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1112
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Recent Articles
1.
Complex equilibrium involving aldosterone underlies the pathophysiology of renovascular hypertension
Adachi M, Fujii T, Ochi A, Ishida T, Motegi S, Nagahara K, et al.
Clin Pediatr Endocrinol
. 2025 Jan;
34(1):19-26.
PMID: 39777133
The mechanisms underlying the maintenance of hypertension in renovascular hypertension (RVH) are not well understood. To test the current concept of RVH pathophysiology, circulating aldosterone levels in clinical cases were...
2.
Motegi S, Adachi M, Nagahara K, Ishida T, Ochi A, Mizuno K
Clin Pediatr Endocrinol
. 2025 Jan;
34(1):27-35.
PMID: 39777125
Autoimmune hypothyroidism is categorized into Hashimoto thyroiditis (HT) and atrophic thyroiditis (AT). Although a consensus exists among Japanese endocrinologists that pediatric AT is associated with severe hypothyroidism, the question remains...
3.
Hayashi M, Narumi S, Sato T, Amano N, Suwanai A, Haruna H, et al.
J Clin Endocrinol Metab
. 2024 Nov;
PMID: 39569443
Context: 46,XY differences of sex development (DSD) are heterogeneous in etiology. The detailed phenotypes of 46,XY DSD patients with monogenic disorders have not been fully elucidated. Objectives: To estimate the...
4.
Toyoda J, Adachi M, Ochi A, Okada Y, Honda A, Mizuno K, et al.
Clin Pediatr Endocrinol
. 2023 Jun;
32(3):155-160.
PMID: 37362166
Pseudo-Bartter syndrome (PBS) develops owing to renal or extrarenal chloride loss, leading to hypokalemic alkalosis. Whereas most adult cases result from diuretic/laxative abuse, many infantile cases occur secondary to cystic...
5.
Adachi M, Motegi S, Nagahara K, Ochi A, Toyoda J, Mizuno K
Endocr J
. 2023 Apr;
70(7):723-729.
PMID: 37081692
Pseudohypoaldosteronism (PHA) type II (PHA2) is a genetic disorder that leads to volume overload and hyperkalemic metabolic acidosis. PHA2 and PHA type I (PHA1) have been considered to be genetic...
6.
Tanase-Nakao K, Muroya K, Adachi M, Abe K, Hasegawa T, Narumi S
Clin Pediatr Endocrinol
. 2022 Nov;
31(4):250-255.
PMID: 36405439
PAX8 is a transcription factor that is expressed in the thyroid gland and kidneys. Monoallelic loss-of-function variants cause congenital hypothyroidism (CH), and urogenital malformations are infrequent complications seen in less...
7.
Adachi M, Nagahara K, Ochi A, Toyoda J, Muroya K, Mizuno K
J Endocr Soc
. 2022 Nov;
6(12):bvac147.
PMID: 36320629
Context: Pseudohypoaldosteronism type 1 (PHA1) has been treated as a genetic variant of type IV renal tubular acidosis (RTA), leading to the conception that PHA1 develops hyperchloremic acidosis with a...
8.
Narumi S, Opitz R, Nagasaki K, Muroya K, Asakura Y, Adachi M, et al.
Hum Mol Genet
. 2022 May;
31(23):3967-3974.
PMID: 35535691
Congenital hypothyroidism due to thyroid dysgenesis (TD), presented as thyroid aplasia, hypoplasia or ectopia, is one of the most prevalent rare diseases with an isolated organ malformation. The pathogenesis of...
9.
Iwano R, Toki M, Hanakawa J, Asakura Y, Adachi M, Tanaka Y, et al.
Biomed Chromatogr
. 2021 Sep;
36(1):e5249.
PMID: 34569083
Thyroid dysfunction is common in patients with Down syndrome (DS), the most common chromosomal disorder. Thyroid hormones (THs) are important for normal growth, neurodevelopment, and metabolism, highlighting the importance of...
10.
Ito N, Kubota T, Kitanaka S, Fujiwara I, Adachi M, Takeuchi Y, et al.
J Bone Miner Metab
. 2021 Jul;
39(6):1066-1075.
PMID: 34255195
Introduction: Measurement of fibroblast growth factor 23 (FGF23) has been reported to be clinically useful for the differential diagnosis of chronic hypophosphatemia. However, assays for research use only are available...