Masamichi Yamamoto
Overview
Explore the profile of Masamichi Yamamoto including associated specialties, affiliations and a list of published articles.
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Articles
47
Citations
935
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Recent Articles
1.
Takahashi M, Yamamoto S, Yamamoto S, Okubo A, Nakagawa Y, Kuwahara K, et al.
Nat Commun
. 2024 Nov;
15(1):9977.
PMID: 39578451
Acute kidney injury (AKI), typically caused by ischemia, is a common clinical complication with a poor prognosis. Although proteinuria is an important prognostic indicator of AKI, the underlying causal mechanism...
2.
Watanuki S, Kobayashi H, Sugiura Y, Yamamoto M, Karigane D, Shiroshita K, et al.
Elife
. 2024 Oct;
13.
PMID: 39436045
No abstract available.
3.
Bourgeois Yoshioka C, Takenaka-Ninagawa N, Goto M, Miki M, Watanabe D, Yamamoto M, et al.
Stem Cell Res Ther
. 2024 Sep;
15(1):313.
PMID: 39300595
Background: Duchenne muscular dystrophy (DMD) is an incurable neuromuscular disease leading to progressive skeletal muscle weakness and fatigue. Cell transplantation in murine models has shown promise in supplementing the lack...
4.
Yamamoto S, Yamamoto S, Takahashi M, Mii A, Okubo A, Toriu N, et al.
Kidney Int
. 2024 Jul;
106(3):470-481.
PMID: 38996810
ATP depletion plays a central role in the pathogenesis of kidney diseases. Recently, we reported spatiotemporal intracellular ATP dynamics during ischemia reperfusion (IR) using GO-ATeam2 mice systemically expressing an ATP...
5.
Watanuki S, Kobayashi H, Sugiura Y, Yamamoto M, Karigane D, Shiroshita K, et al.
Cell Stem Cell
. 2024 May;
31(8):1145-1161.e15.
PMID: 38772377
Aging generally predisposes stem cells to functional decline, impairing tissue homeostasis. Here, we report that hematopoietic stem cells (HSCs) acquire metabolic resilience that promotes cell survival. High-resolution real-time ATP analysis...
6.
Song X, Nihashi Y, Imai Y, Mori N, Kagaya N, Suenaga H, et al.
Int J Mol Sci
. 2024 Apr;
25(7).
PMID: 38612551
Pancreatic ductal adenocarcinoma (PDAC) is a solid-tumor malignancy. To enhance the treatment landscape of PDAC, a 3D model optimized for rigorous drug screening is essential. Within the PDAC tumor microenvironment,...
7.
Watanuki S, Kobayashi H, Sugiura Y, Yamamoto M, Karigane D, Shiroshita K, et al.
Elife
. 2024 Apr;
12.
PMID: 38573813
Metabolic pathways are plastic and rapidly change in response to stress or perturbation. Current metabolic profiling techniques require lysis of many cells, complicating the tracking of metabolic changes over time...
8.
Hayashi R, Okumura H, Isono M, Yamauchi M, Unami D, Lusi R, et al.
J Radiat Res
. 2024 Mar;
65(3):263-271.
PMID: 38461549
Ionizing radiation (IR)-induced double-strand breaks (DSBs) are primarily repaired by non-homologous end joining or homologous recombination (HR) in human cells. DSB repair requires adenosine-5'-triphosphate (ATP) for protein kinase activities in...
9.
Song X, Nihashi Y, Yamamoto M, Setoyama D, Kunisaki Y, Kida Y
Bioengineering (Basel)
. 2023 Dec;
10(12).
PMID: 38136028
Pancreatic ductal adenocarcinoma (PDAC) is a refractory tumor with a poor prognosis, and its complex microenvironment is characterized by a fibrous interstitial matrix surrounding PDAC cells. Type I collagen is...
10.
Nihashi Y, Song X, Yamamoto M, Setoyama D, Kida Y
Int J Mol Sci
. 2023 Jul;
24(13).
PMID: 37446193
Pancreatic ductal adenocarcinoma (PDAC) is a highly aggressive cancer with a poor prognosis, largely due to its unique tumor microenvironment (TME) and dense fibrotic stroma. Cancer-associated fibroblasts (CAFs) play a...