M T Barrett
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Explore the profile of M T Barrett including associated specialties, affiliations and a list of published articles.
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16
Citations
627
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Recent Articles
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Barrett M, Glogovac J, Prevo L, Reid B, Porter P, Rabinovitch P
Biotechniques
. 2002 Apr;
32(4):888-90, 892, 894, 896.
PMID: 11962610
Microarray technologies have made possible comprehensive analyses of nucleic acid sequence and expression. However, the technology to obtain efficiently high-quality RNA and DNA suitable for array analysis from purified populations...
3.
Sweetser D, Chen C, Blomberg A, Flowers D, Galipeau P, Barrett M, et al.
Blood
. 2001 Aug;
98(4):1188-94.
PMID: 11493469
A genome-wide screening for loss of heterozygosity (LOH), a marker for possible involvement of tumor suppressor genes, was conducted in 53 children with de novo acute myelogenous leukemia (AML). A...
4.
Barrett M, Sanchez C, Prevo L, Wong D, Galipeau P, Paulson T, et al.
Nat Genet
. 1999 May;
22(1):106-9.
PMID: 10319873
It has been hypothesized that neoplastic progression develops as a consequence of an acquired genetic instability and the subsequent evolution of clonal populations with accumulated genetic errors. Accordingly, human cancers...
5.
Funk J, Schiller P, Barrett M, Wong D, Kind P, Sander C
J Cutan Pathol
. 1998 Aug;
25(6):291-6.
PMID: 9694617
The product of the p16/INK4a/CDKN2/MTS1 tumor-suppressor gene acts as a negative cell cycle regulator by inhibiting G1 cyclin-dependent kinases that phosphorylate the retinoblastoma protein. p16 is inactivated in a wide...
6.
Foster S, Wong D, Barrett M, Galloway D
Mol Cell Biol
. 1998 Apr;
18(4):1793-801.
PMID: 9528751
Proliferation of human mammary epithelial cells (HMEC) is limited to a few passages in culture due to an arrest in G1 termed selection or mortality stage 0, M0. A small...
7.
Palanca-Wessels M, Barrett M, Galipeau P, Rohrer K, Reid B, Rabinovitch P
Gastroenterology
. 1998 Feb;
114(2):295-304.
PMID: 9453489
Background & Aims: Progression to cancer in Barrett's esophagus occurs through an accumulation of cell cycle and genetic abnormalities that have been documented in vivo. To better study neoplastic evolution...
8.
Wong D, Barrett M, Stoger R, Emond M, Reid B
Cancer Res
. 1997 Jul;
57(13):2619-22.
PMID: 9205067
Loss of heterozygosity (LOH) of 9p21, which contains the p16INK4a tumor suppressor gene locus, is one of the most frequent genetic abnormalities in human neoplasia, including esophageal adenocarcinomas. Only a...
9.
Reid B, Barrett M, Galipeau P, Sanchez C, Neshat K, Cowan D, et al.
Eur J Cancer Prev
. 1996 Dec;
5 Suppl 2:57-65.
PMID: 9061296
No abstract available.
10.
Barrett M, Sanchez C, Galipeau P, Neshat K, Emond M, Reid B
Oncogene
. 1996 Nov;
13(9):1867-73.
PMID: 8934532
High frequency allelic loss of chromosome 9p21 has been reported in a number of human cancers, including those of the esophagus. The CDKN2 gene on chromosome 9p21 that encodes the...