M Isabel Aller
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Explore the profile of M Isabel Aller including associated specialties, affiliations and a list of published articles.
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17
Citations
697
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Recent Articles
1.
Kakegawa W, Paternain A, Matsuda K, Aller M, Iida I, Miura E, et al.
Cell Rep
. 2024 Jul;
43(7):114427.
PMID: 38986610
Kainate (KA)-type glutamate receptors (KARs) are implicated in various neuropsychiatric and neurological disorders through their ionotropic and metabotropic actions. However, compared to AMPA- and NMDA-type receptor functions, many aspects of...
2.
Arora V, Pecoraro V, Aller M, Roman C, Paternain A, Lerma J
Cell Rep
. 2018 Jun;
23(13):3827-3838.
PMID: 29949767
Altered glutamatergic neurotransmission is thought to contribute to mental disorders and neurodegenerative diseases. Copy-number variation in genes associated with glutamatergic synapses represents a source of genetic variability, possibly underlying neurological...
3.
Aller M, Pecoraro V, Paternain A, Canals S, Lerma J
J Neurosci
. 2015 Oct;
35(40):13619-28.
PMID: 26446216
Significance Statement: A genetic overlap exists between autism spectrum disorders (ASD), currently thought to represent a continuum of the same disorder with varying degrees of severity, and other neurodevelopmental and...
4.
Rutkowska-Wlodarczyk I, Aller M, Valbuena S, Bologna J, Prezeau L, Lerma J
J Neurosci
. 2015 Apr;
35(13):5171-9.
PMID: 25834043
Kainate receptors (KARs) are found ubiquitously in the CNS and are present presynaptically and postsynaptically regulating synaptic transmission and excitability. Functional studies have proven that KARs act as ion channels...
5.
Palacios-Filardo J, Aller M, Lerma J
Cereb Cortex
. 2014 Oct;
26(4):1464-72.
PMID: 25316333
When native and recombinant kainate receptors (KARs) are compared, there is a mismatch in several of their functional properties. While both generate currents, synaptic responses mediated by KARs have rarely...
6.
Selak S, Paternain A, Aller M, Aller I, Pico E, Rivera R, et al.
Neuron
. 2009 Aug;
63(3):357-71.
PMID: 19679075
Regulation of surface insertion and internalization of AMPA and NMDA receptors has emerged as a key mechanism for the control of synaptic strength. Regulatory elements for synaptic kainate receptors (KARs)...
7.
Trapp S, Aller M, Wisden W, Gourine A
J Neurosci
. 2008 Aug;
28(35):8844-50.
PMID: 18753386
Acid-sensitive K+ channels of the tandem P-domain K+-channel family (TASK-1 and TASK-3) have been implicated in peripheral and central respiratory chemosensitivity; however, because of the lack of decisive pharmacological agents,...
8.
Linden A, Aller M, Leppa E, Rosenberg P, Wisden W, Korpi E
J Pharmacol Exp Ther
. 2008 Jul;
327(1):277-86.
PMID: 18660435
TASK two-pore-domain leak K(+) channels occur throughout the brain. However, TASK-1 and TASK-3 knockout (KO) mice have few neurological impairments and only mildly reduced sensitivities to inhalational anesthetics, contrasting with...
9.
Heitzmann D, Derand R, Jungbauer S, Bandulik S, Sterner C, Schweda F, et al.
EMBO J
. 2007 Nov;
27(1):179-87.
PMID: 18034154
TASK1 (KCNK3) and TASK3 (KCNK9) are two-pore domain potassium channels highly expressed in adrenal glands. TASK1/TASK3 heterodimers are believed to contribute to the background conductance whose inhibition by angiotensin II...
10.
Linden A, Sandu C, Aller M, Vekovischeva O, Rosenberg P, Wisden W, et al.
J Pharmacol Exp Ther
. 2007 Sep;
323(3):924-34.
PMID: 17875609
The TASK-3 channel is an acid-sensitive two-pore-domain K+ channel, widely expressed in the brain and probably involved in regulating numerous neuronal populations. Here, we characterized the behavioral and pharmacological phenotypes...