Luis F Menezes
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Explore the profile of Luis F Menezes including associated specialties, affiliations and a list of published articles.
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20
Citations
906
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Recent Articles
1.
Lin C, Menezes L, Qiu J, Pearson E, Zhou F, Ishimoto Y, et al.
PLoS One
. 2023 Aug;
18(8):e0289778.
PMID: 37540694
PKD1 is the most commonly mutated gene causing autosomal dominant polycystic kidney disease (ADPKD). It encodes Polycystin-1 (PC1), a putative membrane protein that undergoes a set of incompletely characterized post-transcriptional...
2.
Ishimoto Y, Menezes L, Zhou F, Yoshida T, Komori T, Qiu J, et al.
Kidney Int
. 2023 Jul;
104(3):611-616.
PMID: 37419448
No abstract available.
3.
Qiu J, Germino G, Menezes L
Adv Kidney Dis Health
. 2023 Apr;
30(3):209-219.
PMID: 37088523
Autosomal dominant polycystic kidney disease is the most common inherited cause of end-stage kidney disease worldwide. Most cases result from mutation of either of 2 genes, PKD1 and PKD2, which...
4.
Habraken A, Aksen T, Alves J, Amaral R, Betaieb E, Chandola N, et al.
Int J Mater Form
. 2022 Jul;
15(5):61.
PMID: 35855077
This article details the ESAFORM Benchmark 2021. The deep drawing cup of a 1 mm thick, AA 6016-T4 sheet with a strong cube texture was simulated by 11 teams relying...
5.
Terabayashi T, Menezes L, Zhou F, Cai H, Walter P, Garraffo H, et al.
Kidney360
. 2022 Apr;
2(10):1576-1591.
PMID: 35372986
Background: Multiple studies of tissue and cell samples from patients and preclinical models of autosomal dominant polycystic kidney disease report abnormal mitochondrial function and morphology and suggest metabolic reprogramming is...
6.
Terabayashi T, Germino G, Menezes L
Cell Signal
. 2020 Jul;
74:109701.
PMID: 32649993
Systems-based, agnostic approaches focusing on transcriptomics data have been employed to understand the pathogenesis of polycystic kidney diseases (PKD). While multiple signaling pathways, including Wnt, mTOR and G-protein-coupled receptors, have...
7.
Lin C, Kurashige M, Liu Y, Terabayashi T, Ishimoto Y, Wang T, et al.
Sci Rep
. 2018 Feb;
8(1):2743.
PMID: 29426897
Recent studies have reported intrinsic metabolic reprogramming in Pkd1 knock-out cells, implicating dysregulated cellular metabolism in the pathogenesis of polycystic kidney disease. However, the exact nature of the metabolic changes...
8.
Kaimori J, Lin C, Outeda P, Garcia-Gonzalez M, Menezes L, Hartung E, et al.
Sci Rep
. 2017 Aug;
7(1):7733.
PMID: 28798345
Autosomal recessive polycystic kidney disease (ARPKD) is an important childhood nephropathy, occurring 1 in 20,000 live births. The major clinical phenotypes are expressed in the kidney with dilatation of the...
9.
Menezes L, Lin C, Zhou F, Germino G
EBioMedicine
. 2016 Apr;
5:183-92.
PMID: 27077126
Background: The major gene mutated in autosomal dominant polycystic kidney disease was first identified over 20 years ago, yet its function remains poorly understood. We have used a systems-based approach...
10.
Lee Y, Sante J, Comerci C, Cyge B, Menezes L, Li F, et al.
Mol Biol Cell
. 2014 Aug;
25(19):2919-33.
PMID: 25103236
Defects in centrosome and cilium function are associated with phenotypically related syndromes called ciliopathies. Cby1, the mammalian orthologue of the Drosophila Chibby protein, localizes to mature centrioles, is important for...