Laurence Tiret
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Explore the profile of Laurence Tiret including associated specialties, affiliations and a list of published articles.
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Recent Articles
11.
Bottolo L, Chadeau-Hyam M, Hastie D, Zeller T, Liquet B, Newcombe P, et al.
PLoS Genet
. 2013 Aug;
9(8):e1003657.
PMID: 23950726
Genome-wide association studies (GWAS) yielded significant advances in defining the genetic architecture of complex traits and disease. Still, a major hurdle of GWAS is narrowing down multiple genetic associations to...
12.
Garnier S, Truong V, Brocheton J, Zeller T, Rovital M, Wild P, et al.
PLoS Genet
. 2013 Feb;
9(1):e1003240.
PMID: 23382694
In order to assess whether gene expression variability could be influenced by several SNPs acting in cis, either through additive or more complex haplotype effects, a systematic genome-wide search for...
13.
Verdugo R, Zeller T, Rotival M, Wild P, Munzel T, Lackner K, et al.
PLoS One
. 2013 Feb;
8(1):e50888.
PMID: 23372645
Smoking is a risk factor for atherosclerosis with reported widespread effects on gene expression in circulating blood cells. We hypothesized that a molecular signature mediating the relation between smoking and...
14.
Greliche N, Zeller T, Wild P, Rotival M, Schillert A, Ziegler A, et al.
PLoS One
. 2012 Oct;
7(9):e45863.
PMID: 23029284
We aimed to assess whether pri-miRNA SNPs (miSNPs) could influence monocyte gene expression, either through marginal association or by interacting with polymorphisms located in 3'UTR regions (3utrSNPs). We then conducted...
15.
Germain M, Saut N, Oudot-Mellakh T, Letenneur L, Dupuy A, Bertrand M, et al.
PLoS One
. 2012 Jun;
7(6):e38538.
PMID: 22675575
By applying an imputation strategy based on the 1000 Genomes project to two genome-wide association studies (GWAS), we detected a susceptibility locus for venous thrombosis on chromosome 11p11.2 that was...
16.
Plichart M, Empana J, Lambert J, Amouyel P, Tiret L, Letenneur L, et al.
Atherosclerosis
. 2012 Mar;
222(1):187-90.
PMID: 22436605
Objectives: To address the relationship of rs1333049, the 9p21 variant showing the strongest association with coronary heart disease (CHD), with carotid plaques and plaque-free common carotid artery intima-media thickness (CCA-IMT)...
17.
Statistical colocalization of monocyte gene expression and genetic risk variants for type 1 diabetes
Wallace C, Rotival M, Cooper J, Rice C, Yang J, McNeill M, et al.
Hum Mol Genet
. 2012 Mar;
21(12):2815-24.
PMID: 22403184
One mechanism by which disease-associated DNA variation can alter disease risk is altering gene expression. However, linkage disequilibrium (LD) between variants, mostly single-nucleotide polymorphisms (SNPs), means it is not sufficient...
18.
Rotival M, Zeller T, Wild P, Maouche S, Szymczak S, Schillert A, et al.
PLoS Genet
. 2011 Dec;
7(12):e1002367.
PMID: 22144904
One major expectation from the transcriptome in humans is to characterize the biological basis of associations identified by genome-wide association studies. So far, few cis expression quantitative trait loci (eQTLs)...
19.
Wild P, Schnabel R, Lubos E, Zeller T, Sinning C, Keller T, et al.
Clin Chem
. 2011 Nov;
58(1):226-36.
PMID: 22065157
Background: Midregional proadrenomedullin (MR-proADM) is a newly identified prognostic marker in heart failure. We evaluated the prognostic impact of MR-proADM in a cohort of patients with symptomatic coronary artery disease...
20.
Germain M, Saut N, Greliche N, Dina C, Lambert J, Perret C, et al.
PLoS One
. 2011 Oct;
6(9):e25581.
PMID: 21980494
Background: Venous Thrombosis (VT) is a common multifactorial disease associated with a major public health burden. Genetics factors are known to contribute to the susceptibility of the disease but how...