L Cody Smith
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Explore the profile of L Cody Smith including associated specialties, affiliations and a list of published articles.
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10
Citations
191
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Recent Articles
1.
Venosa A, Smith L, Gow A, Zarbl H, Laskin J, Laskin D
Toxicol Appl Pharmacol
. 2021 May;
423:115569.
PMID: 33971176
Activated macrophages have been implicated in lung injury and fibrosis induced by the cytotoxic alkylating agent, nitrogen mustard (NM). Herein, we determined if macrophage activation is associated with histone modifications...
2.
Chen H, Humes S, Rose M, Robinson S, Loeb J, Sabaraya I, et al.
Toxicol Appl Pharmacol
. 2020 Aug;
404:115167.
PMID: 32771490
Growing use of carbon nanotubes (CNTs) have garnered concerns regarding their association with adverse health effects. Few studies have probed how CNTs affect a host's susceptibility to pathogens, particularly respiratory...
3.
Smith L, Venosa A, Gow A, Laskin J, Laskin D
Ann N Y Acad Sci
. 2020 Aug;
1480(1):146-154.
PMID: 32767459
Nitrogen mustard (NM) and sulfur mustard are cytotoxic alkylating agents that cause severe and progressive damage to the respiratory tract. Evidence indicates that macrophages play a key role in the...
4.
Smith L, Moreno S, Robinson S, Orandle M, Porter D, Das D, et al.
NanoImpact
. 2020 Apr;
14.
PMID: 32313843
Exposure to multi-walled carbon nanotubes (MWCNTs) is suspected to contribute to pulmonary fibrosis through modulation of transforming growth factor beta1 (TGF-β1). There is growing evidence that estrogen signaling is important...
5.
Smith L, Moreno S, Robertson L, Robinson S, Gant K, Bryant A, et al.
Respir Res
. 2018 Sep;
19(1):160.
PMID: 30165855
Background: Sex differences in idiopathic pulmonary fibrosis (IPF) suggest a protective role for estrogen (E2); however, mechanistic studies in animal models have produced mixed results. Reports using cell lines have...
6.
Villeneuve D, Angrish M, Fortin M, Katsiadaki I, Leonard M, Margiotta-Casaluci L, et al.
Environ Toxicol Chem
. 2018 Mar;
37(6):1734-1748.
PMID: 29492998
Toxicological responses to stressors are more complex than the simple one-biological-perturbation to one-adverse-outcome model portrayed by individual adverse outcome pathways (AOPs). Consequently, the AOP framework was designed to facilitate de...
7.
Knapen D, Angrish M, Fortin M, Katsiadaki I, Leonard M, Margiotta-Casaluci L, et al.
Environ Toxicol Chem
. 2018 Mar;
37(6):1723-1733.
PMID: 29488651
Based on the results of a Horizon Scanning exercise sponsored by the Society of Environmental Toxicology and Chemistry that focused on advancing the adverse outcome pathway (AOP) framework, the development...
8.
Humes S, Hentschel S, Lavelle C, Smith L, Lednicky J, Saleh N, et al.
Biotechniques
. 2017 Aug;
63(2):81-84.
PMID: 28803544
Nanomaterials (NMs) of various types, including carbon nanotubes (CNTs), can interfere with standard quantitative real-time PCR (qRT-PCR) assays, resulting in inaccurate gene expression measurements; however, the precise step in the...
9.
Smith L, Clark J, Bisesi Jr J, Ferguson P, Sabo-Attwood T
Comp Biochem Physiol Part D Genomics Proteomics
. 2016 May;
19:159-173.
PMID: 27156127
The diverse biological effects of xenoestrogens may be explained by their ability to differentially recruit co-regulatory proteins to the estrogen receptor (ER). We employed high-throughput receptor affinity binding and co-regulatory...
10.
Smith L, Ralston-Hooper K, Ferguson P, Sabo-Attwood T
Toxicol Sci
. 2016 Mar;
151(2):434-46.
PMID: 27026707
Estrogen exerts cellular effects through both nuclear (ESR1 and ESR2) and membrane-bound estrogen receptors (G-protein coupled estrogen receptor, GPER); however, it is unclear if they act independently or engage in...