Kuniyoshi Fukai
Overview
Explore the profile of Kuniyoshi Fukai including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
14
Citations
348
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
11.
Kaimoto S, Hoshino A, Ariyoshi M, Okawa Y, Tateishi S, Ono K, et al.
Am J Physiol Heart Circ Physiol
. 2016 Dec;
312(2):H305-H313.
PMID: 28011586
Failing heart loses its metabolic flexibility, relying increasingly on glucose as its preferential substrate and decreasing fatty acid oxidation (FAO). Peroxisome proliferator-activated receptor α (PPAR-α) is a key regulator of...
12.
Fukai K, Nakamura A, Hoshino A, Nakanishi N, Okawa Y, Ariyoshi M, et al.
Am J Physiol Heart Circ Physiol
. 2015 Feb;
308(8):H951-9.
PMID: 25659487
Pulmonary arterial hypertension (PAH) is a refractory disease characterized by uncontrolled vascular remodeling and elevated pulmonary arterial pressure. Although synthetic inhibitors of some tyrosine kinases have been used to treat...
13.
Hoshino A, Okawa Y, Ariyoshi M, Kaimoto S, Uchihashi M, Fukai K, et al.
Circ Heart Fail
. 2014 Apr;
7(3):500-9.
PMID: 24740269
Background: Mitochondrial compromise is a fundamental contributor to heart failure. Recent studies have revealed that several surveillance systems maintain mitochondrial integrity. The present study evaluated the role of mitochondrial AAA+...
14.
Hoshino A, Ariyoshi M, Okawa Y, Kaimoto S, Uchihashi M, Fukai K, et al.
Proc Natl Acad Sci U S A
. 2014 Feb;
111(8):3116-21.
PMID: 24516131
Mitochondrial compromise is a fundamental contributor to pancreatic β-cell failure in diabetes. Previous studies have demonstrated a broader role for tumor suppressor p53 that extends to the modulation of mitochondrial...