Koki Aihara
Overview
Explore the profile of Koki Aihara including associated specialties, affiliations and a list of published articles.
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Articles
7
Citations
1025
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0
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Recent Articles
1.
Nomura M, Saito K, Aihara K, Nagae G, Yamamoto S, Tatsuno K, et al.
Sci Rep
. 2019 May;
9(1):7935.
PMID: 31118432
A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has been fixed in the paper.
2.
Nomura M, Saito K, Aihara K, Nagae G, Yamamoto S, Tatsuno K, et al.
Sci Rep
. 2019 Feb;
9(1):1903.
PMID: 30760837
To elucidate the mechanisms of malignant progression of lower-grade glioma, molecular profiling using methylation array, whole-exome sequencing, and RNA sequencing was performed for 122, 36 and 31 gliomas, respectively. This...
3.
Aihara K, Mukasa A, Nagae G, Nomura M, Yamamoto S, Ueda H, et al.
Acta Neuropathol Commun
. 2017 Mar;
5(1):18.
PMID: 28270234
Among diffuse gliomas, oligodendrogliomas show relatively better prognosis, respond well to radiotherapy and chemotherapy, and seldom progress to very aggressive tumors. To elucidate the genetic and epigenetic background for such...
4.
Ikemura M, Shibahara J, Mukasa A, Takayanagi S, Aihara K, Saito N, et al.
Histopathology
. 2016 Jan;
69(2):260-7.
PMID: 26741321
Aims: We performed an immunohistochemical analysis of alpha-thalassaemia/mental retardation syndrome X-linked (ATRX) expression in adult diffuse gliomas, with reference to clinicopathological and genetic features, to determine the utility of this...
5.
van Thuijl H, Mazor T, Johnson B, Fouse S, Aihara K, Hong C, et al.
Acta Neuropathol
. 2015 Mar;
129(4):597-607.
PMID: 25724300
Temozolomide (TMZ) increases the overall survival of patients with glioblastoma (GBM), but its role in the clinical management of diffuse low-grade gliomas (LGG) is still being defined. DNA hypermethylation of...
6.
Johnson B, Mazor T, Hong C, Barnes M, Aihara K, McLean C, et al.
Science
. 2013 Dec;
343(6167):189-193.
PMID: 24336570
Tumor recurrence is a leading cause of cancer mortality. Therapies for recurrent disease may fail, at least in part, because the genomic alterations driving the growth of recurrences are distinct...
7.
Aihara K, Mukasa A, Gotoh K, Saito K, Nagae G, Tsuji S, et al.
Neuro Oncol
. 2013 Nov;
16(1):140-6.
PMID: 24285547
Introduction: Mutations in H3F3A, which encodes histone H3.3, commonly occur in pediatric glioblastoma. Additionally, H3F3A K27M substitutions occur in gliomas that arise at midline locations (eg, pons, thalamus, spine); moreover,...