Kimber L Converso
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Explore the profile of Kimber L Converso including associated specialties, affiliations and a list of published articles.
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6
Citations
239
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Recent Articles
1.
Palpant N, Day S, Herron T, Converso K, Metzger J
Cardiovasc Res
. 2008 Jul;
80(2):209-18.
PMID: 18635554
Aims: Contractile dysfunction associated with myocardial ischaemia is a significant cause of morbidity and mortality in the elderly. Strategies to protect the aged heart from ischaemia-mediated pump failure are needed....
2.
Day S, Coutu P, Wang W, Herron T, Turner I, Shillingford M, et al.
Physiol Genomics
. 2008 Mar;
33(3):312-22.
PMID: 18334547
Relaxation abnormalities are prevalent in heart failure and contribute to clinical outcomes. Disruption of Ca2+ homeostasis in heart failure delays relaxation by prolonging the intracellular Ca2+ transient. We sought to...
3.
Kasahara H, Ueyama T, Wakimoto H, Liu M, Maguire C, Converso K, et al.
J Mol Cell Cardiol
. 2003 Apr;
35(3):243-56.
PMID: 12676539
Nkx2.5, an evolutionarily conserved homeodomain containing transcription factor, is one of the earliest cardiogenic markers. Although its expression continues through adulthood, its function in adult cardiomyocytes is not well understood....
4.
Min J, Yang Y, Sullivan M, Ke Q, Converso K, Chen Y, et al.
J Thorac Cardiovasc Surg
. 2003 Feb;
125(2):361-9.
PMID: 12579106
Objective: This study was designed to investigate the feasibility of and potential functional improvement with embryonic stem cell transplantation in rats 32 weeks after myocardial infarction. Methods: Before cell transplantation,...
5.
Min J, Sullivan M, Yang Y, Zhang J, Converso K, Morgan J, et al.
Ann Thorac Surg
. 2002 Nov;
74(5):1568-75.
PMID: 12440610
Background: Viable cardiomyocytes after myocardial infarction (MI) are unable to repair the necrotic myocardium due to their limited capability of regeneration. The present study investigated whether intramyocardial transplantation of human...
6.
Min J, Yang Y, Converso K, Liu L, Huang Q, Morgan J, et al.
J Appl Physiol (1985)
. 2001 Dec;
92(1):288-96.
PMID: 11744672
Massive loss of cardiac myocytes after myocardial infarction (MI) is a common cause of heart failure. The present study was designed to investigate the improvement of cardiac function in MI...