Kevin K Frick
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Explore the profile of Kevin K Frick including associated specialties, affiliations and a list of published articles.
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17
Citations
326
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Recent Articles
1.
Krieger N, Grynpas M, VandenEynde A, Asplin J, Frick K, Kim M, et al.
Nephron
. 2019 Feb;
142(2):147-158.
PMID: 30726853
Background: Urine (u) calcium (Ca) excretion is directly dependent on dietary sodium (Na) intake leading to the recommendation for Na restriction in hypercalciuric kidney stone formers. However, there is no...
2.
Frick K, Krieger N, Bushinsky D
Curr Opin Nephrol Hypertens
. 2015 Jun;
24(4):336-44.
PMID: 26050120
Purpose Of Review: In this review, we discuss how the genetic hypercalciuric stone-forming (GHS) rats, which closely model idiopathic hypercalciuria and stone formation in humans, provide insights into the pathophysiology...
3.
Krieger N, Asplin J, Frick K, Granja I, Culbertson C, Ng A, et al.
J Am Soc Nephrol
. 2015 Apr;
26(12):3001-8.
PMID: 25855777
Potassium citrate is prescribed to decrease stone recurrence in patients with calcium nephrolithiasis. Citrate binds intestinal and urine calcium and increases urine pH. Citrate, metabolized to bicarbonate, should decrease calcium...
4.
Frick K, Asplin J, Culbertson C, Granja I, Krieger N, Bushinsky D
Am J Physiol Renal Physiol
. 2014 Feb;
306(9):F1081-7.
PMID: 24573387
Genetic hypercalciuric stone-forming (GHS) rats demonstrate increased intestinal Ca absorption, increased bone resorption, and reduced renal tubular Ca reabsorption leading to hypercalciuria and all form kidney stones. GHS have increased...
5.
Ng A, Frick K, Krieger N, Asplin J, Cohen-McFarlane M, Culbertson C, et al.
Calcif Tissue Int
. 2014 Feb;
94(5):531-43.
PMID: 24481706
Genetic hypercalciuric stone-forming (GHS) rats, bred to maximize urine (u) calcium (Ca) excretion, demonstrate increased intestinal Ca absorption, increased bone Ca resorption, and reduced renal Ca reabsorption, all leading to...
6.
Frick K, Asplin J, Krieger N, Culbertson C, Asplin D, Bushinsky D
Am J Physiol Renal Physiol
. 2013 Aug;
305(8):F1132-8.
PMID: 23926184
The inbred genetic hypercalciuric stone-forming (GHS) rats exhibit many features of human idiopathic hypercalciuria and have elevated levels of vitamin D receptors (VDR) in calcium (Ca)-transporting organs. On a normal-Ca...
7.
Frick K, Asplin J, Favus M, Culbertson C, Krieger N, Bushinsky D
Am J Physiol Renal Physiol
. 2013 Jan;
304(6):F718-26.
PMID: 23344574
Genetic hypercalciuric stone-forming (GHS) rats, bred to maximize urine (U) calcium (Ca) excretion, have increased intestinal Ca absorption and bone Ca resorption and reduced renal Ca reabsorption, leading to increased...
8.
Frick K, Bushinsky D
Am J Physiol Renal Physiol
. 2010 May;
299(2):F418-25.
PMID: 20504884
In vivo, metabolic acidosis {decreased pH from decreased bicarbonate concentration ([HCO(3)(-)])} increases urine calcium (Ca) without increased intestinal Ca absorption, resulting in a loss of bone Ca. Conversely, respiratory acidosis...
9.
Frick K, Krieger N, Nehrke K, Bushinsky D
J Bone Miner Res
. 2008 Oct;
24(2):305-13.
PMID: 18847331
Metabolic acidosis increases urine Ca without increasing intestinal absorption, leading to bone Ca loss. It is unclear how bone cells detect the increase in proton concentration. To determine which G...
10.
Krieger N, Frick K, Strutz K, Michalenka A, Bushinsky D
J Bone Miner Res
. 2007 Mar;
22(6):907-17.
PMID: 17352658
Unlabelled: Chronic metabolic acidosis induces net Ca efflux from bone; this osteoclastic bone resorption is mediated by increased osteoblastic prostaglandin synthesis. Cyclooxygenase, the rate-limiting enzyme in prostaglandin synthesis, is present...