Keri L Janesko-Feldman
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Explore the profile of Keri L Janesko-Feldman including associated specialties, affiliations and a list of published articles.
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12
Citations
304
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Recent Articles
1.
Herrmann J, Jackson T, Fabio A, Clark R, Berger R, Janesko-Feldman K, et al.
J Am Heart Assoc
. 2023 Jul;
12(15):e029972.
PMID: 37489743
No abstract available.
2.
Zusman B, Dixon C, Jha R, Vagni V, Henchir J, Carlson S, et al.
J Neurotrauma
. 2021 Jul;
38(20):2907-2917.
PMID: 34269621
Civilian traumatic brain injury (TBI) guidelines recommend resuscitation of patients with hypotensive TBI with crystalloids. Increasing evidence, however, suggests that whole blood (WB) resuscitation may improve physiological and survival outcomes...
3.
Zusman B, Kochanek P, Bailey Z, Leung L, Vagni V, Okonkwo D, et al.
Neurocrit Care
. 2020 Sep;
34(3):781-794.
PMID: 32886294
Background: Despite increasing use in hemorrhagic shock (HS), whole blood (WB) resuscitation for polytrauma with traumatic brain injury (TBI) is largely unexplored. Current TBI guidelines recommend crystalloid for prehospital resuscitation....
4.
Jha R, Molyneaux B, Jackson T, Wallisch J, Park S, Poloyac S, et al.
J Neurotrauma
. 2018 Apr;
35(17):2125-2135.
PMID: 29648981
Cerebral edema is critical to morbidity/mortality in traumatic brain injury (TBI) and is worsened by hypotension. Glibenclamide may reduce cerebral edema by inhibiting sulfonylurea receptor-1 (Sur1); its effect on diffuse...
5.
Jha R, Puccio A, Chou S, Chang C, Wallisch J, Molyneaux B, et al.
Crit Care Med
. 2016 Nov;
45(3):e255-e264.
PMID: 27845954
Objectives: Cerebral edema is a key poor prognosticator in traumatic brain injury. There are no biomarkers identifying patients at-risk, or guiding mechanistically-precise therapies. Sulfonylurea receptor-1-transient receptor potential cation channel M4...
6.
Shein S, Shellington D, Exo J, Jackson T, Wisniewski S, Jackson E, et al.
J Neurotrauma
. 2014 Apr;
31(16):1386-95.
PMID: 24773520
Secondary insults, such as hemorrhagic shock (HS), worsen outcome from traumatic brain injury (TBI). Both TBI and HS modulate levels of inflammatory mediators. We evaluated the addition of HS on...
7.
Su E, Bell M, Wisniewski S, Adelson P, Janesko-Feldman K, Salonia R, et al.
Dev Neurosci
. 2010 Dec;
32(5-6):385-95.
PMID: 21124000
α-Synuclein is one of the most abundant proteins in presynaptic terminals. Normal expression of α-synuclein is essential for neuronal survival and it prevents the initiation of apoptosis in neurons through...
8.
Foley L, Hitchens T, Ho C, Janesko-Feldman K, Melick J, Bayir H, et al.
J Neurotrauma
. 2009 Aug;
26(9):1509-19.
PMID: 19663686
Macrophages contribute to secondary damage and repair after central nervous system (CNS) injury. Micron-sized paramagnetic iron oxide (MPIO) particles can label macrophages in situ, facilitating three-dimensional (3D) mapping of macrophage...
9.
Bayir H, Adelson P, Wisniewski S, Shore P, Lai Y, Brown D, et al.
Crit Care Med
. 2008 Dec;
37(2):689-95.
PMID: 19114918
Objective: Oxidative stress contributes to secondary damage after traumatic brain injury (TBI). Hypothermia decreases endogenous antioxidant consumption and lipid peroxidation after experimental cerebral injury. Our objective was to determine the...
10.
Bayir H, Tyurin V, Tyurina Y, Viner R, Ritov V, Amoscato A, et al.
Ann Neurol
. 2007 Aug;
62(2):154-69.
PMID: 17685468
Objective: Enhanced lipid peroxidation is well established in traumatic brain injury. However, its molecular targets, identity of peroxidized phospholipid species, and their signaling role have not been deciphered. Methods: Using...