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Katare Gopalrao Rajesh

Explore the profile of Katare Gopalrao Rajesh including associated specialties, affiliations and a list of published articles. Areas
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Articles 8
Citations 134
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Recent Articles
1.
Rajesh K, Suzuki R, Maeda H, Murio Y, Sasaguri S
Life Sci . 2006 Aug; 79(18):1749-55. PMID: 16876202
Even though reperfusion is the treatment of choice in patients admitted with acute myocardial infarction, reperfusion itself has been demonstrated to activate various pathological factors especially following procedures of cardiac...
2.
Rajesh K, Suzuki R, Maeda H, Yamamoto M, Yutong X, Sasaguri S
J Mol Cell Cardiol . 2005 Sep; 39(5):766-76. PMID: 16171810
The opening of mitochondrial permeability transition pore (PTP) during reperfusion injury of heart has been well demonstrated and thus controlling PTP would attenuate the myocardial damage and cell death. Ursodeoxycholic...
3.
Xing Y, Rajesh K, Suzuki R, Yamamoto M, Rao P, Sasaguri S
J Surg Res . 2005 Jun; 127(2):144-50. PMID: 15936033
Background: OG-VI, a well-balanced mixture of nucleoside and nucleotides, has been demonstrated to have a favorable effect on energy metabolism. In this study, we tested the hypothesis that addition of...
4.
Rajesh K, Sasaguri S, Suzuki R, Xing Y, Maeda H
Int J Cardiol . 2004 Jun; 96(1):41-9. PMID: 15203260
Background: Cardiac hypertrophy has been demonstrated to decreases the ATP-sensitive potassium channels (K(ATP)), the major protective mechanism following the energy depletion, a common condition seen during the reperfusion after open...
5.
Rajesh K, Sasaguri S, Ryoko S, Maeda H
Transplantation . 2003 Nov; 76(9):1314-20. PMID: 14627909
Objectives: Long-time preservation of the donor heart before transplantation is associated with mitochondrial damages resulting in functional deterioration after transplantation, although the exact mechanism behind this is still uncertain. Here,...
6.
Rajesh K, Sasaguri S, Zhitian Z, Suzuki R, Asakai R, Maeda H
Cardiovasc Res . 2003 Aug; 59(2):297-307. PMID: 12909313
Objective: The second window of protection (SWOP) following brief coronary artery occlusion begins at 24 h and may last up to 72 h and occurs via many unknown mechanisms. We...
7.
Rajesh K, Sasaguri S, Suzuki R, Maeda H
Am J Physiol Heart Circ Physiol . 2003 Jun; 285(5):H2171-8. PMID: 12816747
Reperfusion after a period of ischemia is associated with the formation of reactive oxygen species (ROS) and Ca2+ overload resulting in the opening of a nonspecific pore in the inner...
8.
Zou Z, Sasaguri S, Rajesh K, Suzuki R
Am J Physiol Heart Circ Physiol . 2002 Oct; 283(5):H1968-74. PMID: 12384475
To investigate the role of high concentrations of dl-3-hydroxybutyrate (DL-3-HB) in preventing heart damage after prolonged fasting, infarct size and the incidence of apoptosis caused by ischemia-reperfusion were determined in...