Joseph S Bednash
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Explore the profile of Joseph S Bednash including associated specialties, affiliations and a list of published articles.
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37
Citations
559
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Recent Articles
1.
Kumar N, Mattoo S, Sanghvi S, Ellendula M, Mahajan S, Planner C, et al.
bioRxiv
. 2024 Dec;
PMID: 39651123
is a gram-negative, opportunistic bacterium abundantly present in the environment. Often infections cause severe pneumonia, if left untreated. Surprisingly, up to 30% of patients admitted to the hospital for community-...
2.
Li P, Faraone J, Hsu C, Chamblee M, Liu Y, Zheng Y, et al.
bioRxiv
. 2024 Nov;
PMID: 39605558
SARS-CoV-2 continues to evolve, producing new variants that drive global COVID-19 surges. XEC, a recombinant of KS.1.1 and KP.3.3, contains T22N and F59S mutations in the spike protein's N-terminal domain...
3.
Li P, Faraone J, Hsu C, Chamblee M, Liu Y, Zheng Y, et al.
bioRxiv
. 2024 Sep;
PMID: 39282390
During the summer of 2024, COVID-19 cases surged globally, driven by variants derived from JN.1 subvariants of SARS-CoV-2 that feature new mutations, particularly in the N-terminal domain (NTD) of the...
4.
Eltobgy M, Johns F, Farkas D, Leuenberger L, Cohen S, Ho K, et al.
Front Immunol
. 2024 Aug;
15:1397629.
PMID: 39161760
Introduction: The acute respiratory distress syndrome (ARDS) is a common complication of severe COVID-19 and contributes to patient morbidity and mortality. ARDS is a heterogeneous syndrome caused by various insults,...
5.
Johnson B, Farkas D, El-Mergawy R, Adair J, Elhance A, Eltobgy M, et al.
Nat Commun
. 2024 Jul;
15(1):6172.
PMID: 39039092
The severity of bacterial pneumonia can be worsened by impaired innate immunity resulting in ineffective pathogen clearance. We describe a mitochondrial protein, aspartyl-tRNA synthetase (DARS2), which is released in circulation...
6.
Li P, Faraone J, Hsu C, Chamblee M, Zheng Y, Carlin C, et al.
Cell Rep
. 2024 Jul;
43(8):114520.
PMID: 39024099
We investigate JN.1-derived subvariants SLip, FLiRT, and KP.2 for neutralization by antibodies in vaccinated individuals, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)-infected patients, or class III monoclonal antibody S309. Compared...
7.
Gunasena M, Alles M, Wijewantha Y, Mulhern W, Bowman E, Gabriel J, et al.
Arterioscler Thromb Vasc Biol
. 2024 Jul;
44(10):e243-e261.
PMID: 38989579
Background: Evidence suggests that COVID-19 predisposes to cardiovascular diseases (CVDs). While monocytes/macrophages play a central role in the immunopathogenesis of atherosclerosis, less is known about their immunopathogenic mechanisms that lead...
8.
Li P, Faraone J, Hsu C, Chamblee M, Zheng Y, Carlin C, et al.
bioRxiv
. 2024 Jun;
PMID: 38826376
SARS-CoV-2 variants derived from the immune evasive JN.1 are on the rise worldwide. Here, we investigated JN.1-derived subvariants SLip, FLiRT, and KP.2 for their ability to be neutralized by antibodies...
9.
Pannu S, Exline M, Bednash J, Englert J, Diaz P, Bartlett A, et al.
Trials
. 2024 May;
25(1):328.
PMID: 38760804
Background: The SARS CoV-2 pandemic has resulted in more than 1.1 million deaths in the USA alone. Therapeutic options for critically ill patients with COVID-19 are limited. Prior studies showed...
10.
Li P, Liu Y, Faraone J, Hsu C, Chamblee M, Zheng Y, et al.
mBio
. 2024 Apr;
15(5):e0075124.
PMID: 38591890
Importance: This study investigates the recently emerged SARS-CoV-2 variants, BA.2.87.1 and JN.1, in comparison to earlier variants and the parental D614G. Varied infectivity and cell-cell fusion activity among these variants...