John E Wagner
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Explore the profile of John E Wagner including associated specialties, affiliations and a list of published articles.
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232
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9732
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Recent Articles
1.
Keahi D, Sanders M, Paul M, Webster A, Fang Y, Wiley T, et al.
bioRxiv
. 2024 Aug;
PMID: 39091814
Biallelic pathogenic variants in the essential DNA repair gene causes Fanconi anemia, complementation group FA-D1. Patients in this group are highly prone to develop embryonal tumors, most commonly medulloblastoma arising...
2.
Bartlett A, Wagner J, Jones B, Wells S, Sabulski A, Fuller C, et al.
Blood Adv
. 2024 Mar;
8(12):3027-3037.
PMID: 38522093
Fanconi anemia (FA) is a complex inherited bone marrow failure syndrome characterized by chromosomal instability and defective DNA repair, causing sensitivity to DNA interstrand crosslinking agents. Our understanding of the...
3.
Saha A, Palchaudhuri R, Lanieri L, Hyzy S, Riddle M, Panthera J, et al.
Blood
. 2024 Mar;
143(21):2201-2216.
PMID: 38447038
Fanconi anemia (FA) is an inherited DNA repair disorder characterized by bone marrow (BM) failure, developmental abnormalities, myelodysplasia, leukemia, and solid tumor predisposition. Allogeneic hematopoietic stem cell transplantation (allo-HSCT), a...
4.
Barbus C, Rayannavar A, Miller B, Jenkins M, Addo O, Rayes A, et al.
Am J Med Genet A
. 2024 Feb;
194(7):e63554.
PMID: 38317562
Patients with Fanconi anemia (FA) are often perceived to have poor growth when general population growth curves are utilized. We hypothesize that FA patients have unique growth and aimed to...
5.
Ramanagoudr-Bhojappa R, Tryon R, Lach F, Donovan F, Maxwell R, Rosenberg A, et al.
Blood Adv
. 2024 Jan;
8(4):899-908.
PMID: 38191666
Fanconi anemia (FA) is a hereditary, DNA repair deficiency disorder caused by pathogenic variants in any 1 of 22 known genes (FANCA-FANCW). Variants in FANCA account for nearly two-thirds of...
6.
Jurdi N, Hoover A, OLeary D, Cao Q, Gupta A, Ebens C, et al.
Transplant Cell Ther
. 2023 Jun;
29(9):576.e1-576.e5.
PMID: 37311510
Graft-versus-host disease (GVHD) is the major toxicity of allogeneic hematopoietic cell transplantation (HCT). We hypothesized that a GVHD prophylaxis regimen of post-transplantation cyclophosphamide (PTCy), tacrolimus (Tac), and mycophenolate mofetil (MMF)...
7.
Holtan S, Hoeschen A, Cao Q, Ustun C, Betts B, Jurdi N, et al.
Transplant Cell Ther
. 2023 Jun;
29(8):509.e1-509.e8.
PMID: 37279855
Treatments that aid inflammation resolution, immune tolerance, and epithelial repair may improve outcomes beyond high-dose corticosteroids and other broad immunosuppressants for life-threatening acute graft-versus-host disease (aGVHD). We studied the addition...
8.
Ramos T, Bolivar-Wagers S, Jin S, Thangavelu G, Simonetta F, Lin P, et al.
Blood
. 2022 Dec;
141(11):1337-1352.
PMID: 36564052
Allogeneic hematopoietic stem cell transplantation (allo-HSCT) is a curative option for patients with hematological disorders and bone marrow (BM) failure syndromes. Graft-versus-host disease (GVHD) remains a leading cause of morbidity...
9.
Webster A, Sanders M, Patel K, Dietrich R, Noonan R, Lach F, et al.
Nature
. 2022 Nov;
612(7940):495-502.
PMID: 36450981
Fanconi anaemia (FA), a model syndrome of genome instability, is caused by a deficiency in DNA interstrand crosslink repair resulting in chromosome breakage. The FA repair pathway protects against endogenous...
10.
Sipe C, Kluesner M, Bingea S, Lahr W, Andrew A, Wang M, et al.
Int J Mol Sci
. 2022 Aug;
23(15).
PMID: 35955545
Fanconi anemia (FA) is a rare genetic disease in which genes essential for DNA repair are mutated. Both the interstrand crosslink (ICL) and double-strand break (DSB) repair pathways are disrupted...