Joelle Vermeulen
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Explore the profile of Joelle Vermeulen including associated specialties, affiliations and a list of published articles.
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21
Citations
983
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Recent Articles
11.
De Preter K, Vermeulen J, Brors B, Delattre O, Eggert A, Fischer M, et al.
Clin Cancer Res
. 2010 Feb;
16(5):1532-41.
PMID: 20179214
Purpose: Reliable prognostic stratification remains a challenge for cancer patients, especially for diseases with variable clinical course such as neuroblastoma. Although numerous studies have shown that outcome might be predicted...
12.
RNA pre-amplification enables large-scale RT-qPCR gene-expression studies on limiting sample amounts
Vermeulen J, Derveaux S, Lefever S, Smet E, De Preter K, Yigit N, et al.
BMC Res Notes
. 2009 Nov;
2:235.
PMID: 19930725
Background: The quantitative polymerase chain reaction (qPCR) is a widely utilized method for gene-expression analysis. However, insufficient material often compromises large-scale gene-expression studies. The aim of this study is to...
13.
Vermeulen J, De Preter K, Laureys G, Speleman F, Vandesompele J
Lancet Oncol
. 2009 Nov;
10(11):1030.
PMID: 19880060
No abstract available.
14.
Vermeulen J, Pattyn F, De Preter K, Vercruysse L, Derveaux S, Mestdagh P, et al.
Nucleic Acids Res
. 2009 Sep;
37(21):e138.
PMID: 19734345
The quantitative polymerase chain reaction (qPCR) is widely utilized for gene expression analysis. However, the lack of robust strategies for cross laboratory data comparison hinders the ability to collaborate or...
15.
Van Roy N, De Preter K, Hoebeeck J, Van Maerken T, Pattyn F, Mestdagh P, et al.
Genome Med
. 2009 Jul;
1(7):74.
PMID: 19638189
Neuroblastoma is one of the most common solid tumors of childhood, arising from immature sympathetic nervous system cells. The clinical course of patients with neuroblastoma is highly variable, ranging from...
16.
Vermeulen J, De Preter K, Naranjo A, Vercruysse L, Van Roy N, Hellemans J, et al.
Lancet Oncol
. 2009 Jun;
10(7):663-71.
PMID: 19515614
Background: More accurate prognostic assessment of patients with neuroblastoma is required to better inform the choice of risk-related therapy. The aim of this study is to develop and validate a...
17.
Janoueix-Lerosey I, Schleiermacher G, Michels E, Mosseri V, Ribeiro A, Lequin D, et al.
J Clin Oncol
. 2009 Jan;
27(7):1026-33.
PMID: 19171713
Purpose: For a comprehensive overview of the genetic alterations of neuroblastoma, their association and clinical significance, we conducted a whole-genome DNA copy number analysis. Patients And Methods: A series of...
18.
Hoebeeck J, Michels E, Pattyn F, Combaret V, Vermeulen J, Yigit N, et al.
Cancer Lett
. 2008 Sep;
273(2):336-46.
PMID: 18819746
CpG island hypermethylation has been recognized as an alternative mechanism for tumor suppressor gene inactivation. In this study, we performed methylation-specific PCR (MSP) to investigate the methylation status of 10...
19.
Michels E, Vandesompele J, De Preter K, Hoebeeck J, Vermeulen J, Schramm A, et al.
Genes Chromosomes Cancer
. 2007 Sep;
46(12):1098-108.
PMID: 17823929
High-resolution array comparative genomic hybridization (arrayCGH) profiling was performed on 75 primary tumors and 29 cell lines to gain further insight into the genetic heterogeneity of neuroblastoma and to refine...
20.
Van Maerken T, Speleman F, Vermeulen J, Lambertz I, De Clercq S, Smet E, et al.
Cancer Res
. 2006 Oct;
66(19):9646-55.
PMID: 17018622
Circumvention of the p53 tumor suppressor barrier in neuroblastoma is rarely caused by TP53 mutation but might arise from inappropriately increased activity of its principal negative regulator MDM2. We show...