Jessica G C A M Buijs-Gladdines
Overview
Explore the profile of Jessica G C A M Buijs-Gladdines including associated specialties, affiliations and a list of published articles.
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12
Citations
680
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Recent Articles
1.
Poort V, Hagelaar R, van Roosmalen M, Trabut L, Buijs-Gladdines J, van Wijk B, et al.
Cancer Res
. 2024 Jun;
84(16):2720-2733.
PMID: 38885294
Leukemia is characterized by oncogenic lesions that result in a block of differentiation, whereas phenotypic plasticity is retained. A better understanding of how these two phenomena arise during leukemogenesis in...
2.
van Hulst A, Van der Zwet J, Buijs-Gladdines J, Smits W, Fiocco M, Pieters R, et al.
Haematologica
. 2024 Jan;
109(5):1551-1556.
PMID: 38205521
No abstract available.
3.
Smits W, Vermeulen C, Hagelaar R, Kimura S, Vroegindeweij E, Buijs-Gladdines J, et al.
Cell Rep
. 2023 Apr;
42(4):112373.
PMID: 37060567
Monoallelic inactivation of CCCTC-binding factor (CTCF) in human cancer drives altered methylated genomic states, altered CTCF occupancy at promoter and enhancer regions, and deregulated global gene expression. In patients with...
4.
Van der Zwet J, Cordo V, Buijs-Gladdines J, Hagelaar R, Smits W, Vroegindeweij E, et al.
Haematologica
. 2022 Jun;
108(3):732-746.
PMID: 35734930
Physiological and pathogenic interleukin-7-receptor (IL7R)-induced signaling provokes glucocorticoid resistance in a subset of patients with pediatric T-cell acute lymphoblastic leukemia (T-ALL). Activation of downstream STAT5 has been suggested to cause...
5.
Van der Zwet J, Buijs-Gladdines J, Cordo V, Debets D, Smits W, Chen Z, et al.
Leukemia
. 2021 May;
35(12):3394-3405.
PMID: 34007050
(Patho-)physiological activation of the IL7-receptor (IL7R) signaling contributes to steroid resistance in pediatric T-cell acute lymphoblastic leukemia (T-ALL). Here, we show that activating IL7R pathway mutations and physiological IL7R signaling...
6.
Van der Zwet J, Smits W, Buijs-Gladdines J, Pieters R, Meijerink J
Hemasphere
. 2020 Dec;
5(1):e513.
PMID: 33364552
The glucocorticoid receptor NR3C1 is essential for steroid-induced apoptosis, and deletions of this gene have been recurrently identified at disease relapse for acute lymphoblastic leukemia (ALL) patients. Here, we demonstrate...
7.
Li Y, Buijs-Gladdines J, Cante-Barrett K, Stubbs A, Vroegindeweij E, Smits W, et al.
PLoS Med
. 2016 Dec;
13(12):e1002200.
PMID: 27997540
Background: Pediatric acute lymphoblastic leukemia (ALL) is the most common childhood cancer and the leading cause of cancer-related mortality in children. T cell ALL (T-ALL) represents about 15% of pediatric...
8.
Mendes R, Sarmento L, Cante-Barrett K, Zuurbier L, Buijs-Gladdines J, Povoa V, et al.
Blood
. 2014 Jun;
124(4):567-78.
PMID: 24904117
Phosphatase and tensin homolog (PTEN)-inactivating mutations and/or deletions are an independent risk factor for relapse of T-cell acute lymphoblastic leukemia (T-ALL) patients treated on Dutch Childhood Oncology Group or German...
9.
Zuurbier L, Gutierrez A, Mullighan C, Cante-Barrett K, Gevaert A, de Rooi J, et al.
Haematologica
. 2013 Aug;
99(1):94-102.
PMID: 23975177
Three distinct immature T-cell acute lymphoblastic leukemia entities have been described including cases that express an early T-cell precursor immunophenotype or expression profile, immature MEF2C-dysregulated T-cell acute lymphoblastic leukemia cluster...
10.
Zuurbier L, Petricoin 3rd E, Vuerhard M, Calvert V, Kooi C, Buijs-Gladdines J, et al.
Haematologica
. 2012 Apr;
97(9):1405-13.
PMID: 22491738
Background: PI3K/AKT pathway mutations are found in T-cell acute lymphoblastic leukemia, but their overall impact and associations with other genetic aberrations is unknown. PTEN mutations have been proposed as secondary...