Jennifer Kistermann
Overview
Explore the profile of Jennifer Kistermann including associated specialties, affiliations and a list of published articles.
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Articles
5
Citations
35
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Recent Articles
1.
Rose M, Huth S, Wiesehofer M, Ehling J, Henkel C, Steitz J, et al.
Cancers (Basel)
. 2022 Feb;
14(3).
PMID: 35158755
Oncogenic drivers such as mutated are the preferred targets in modern drug development. However, restoring the lost function of tumor suppressor proteins could also be a valid approach to combatting...
2.
Marx G, Greiner W, Juhra C, Elkenkamp S, Gensorowsky D, Lemmen S, et al.
J Med Internet Res
. 2022 Feb;
24(3):e34098.
PMID: 35103604
Background: Evidence-based infectious disease and intensive care management is more relevant than ever. Medical expertise in the two disciplines is often geographically limited to university institutions. In addition, the interconnection...
3.
Marx G, Bickenbach J, Fritsch S, Kunze J, Maassen O, Deffge S, et al.
BMJ Open
. 2021 Sep;
11(4):e045589.
PMID: 34550901
Introduction: The acute respiratory distress syndrome (ARDS) is a highly relevant entity in critical care with mortality rates of 40%. Despite extensive scientific efforts, outcome-relevant therapeutic measures are still insufficiently...
4.
Rose M, Noetzel E, Kistermann J, Eschenbruch J, Rushrush S, Gan L, et al.
Cells
. 2021 Apr;
10(5).
PMID: 33924987
This study aims at characterizing the role of the putative tumor suppressor ITIH5 in basal-type bladder cancers (BLCA). By sub-classifying TCGA BLCA data, we revealed predominant loss of ITIH5 expression...
5.
Reetz K, Hilgers R, Isfort S, Dohmen M, Didszun C, Fedosov K, et al.
Neurol Res Pract
. 2020 Dec;
1:33.
PMID: 33324899
Introduction: Currently, no treatment that delays with the progression of Friedreich ataxia is available. In the majority of patients Friedreich ataxia is caused by homozygous pathological expansion of GAA repeats...