Jaslyn Johnson
Overview
Explore the profile of Jaslyn Johnson including associated specialties, affiliations and a list of published articles.
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Articles
12
Citations
199
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Recent Articles
1.
Yang Y, Johnson J, Troupes C, Feldsott E, Kraus L, Megill E, et al.
Redox Biol
. 2023 Oct;
67:102909.
PMID: 37801856
Few therapies have produced significant improvement in cardiac structure and function after ischemic cardiac injury (ICI). Our possible explanation is activation of local inflammatory responses negatively impact the cardiac repair...
2.
Johnson J, Yang Y, Bian Z, Schena G, Li Y, Zhang X, et al.
Circ Res
. 2023 Feb;
132(6):723-740.
PMID: 36799218
Background: A recent study suggests that systemic hypoxemia in adult male mice can induce cardiac myocytes to proliferate. The goal of the present experiments was to confirm these results, provide...
3.
Oliveira Carvalho Rigaud V, Hoy R, Kurian J, Zarka C, Behanan M, Brosious I, et al.
Circulation
. 2022 Oct;
147(4):324-337.
PMID: 36314132
Background: Developmental cardiac tissue holds remarkable capacity to regenerate after injury and consists of regenerative mononuclear diploid cardiomyocytes. On maturation, mononuclear diploid cardiomyocytes become binucleated or polyploid and exit the...
4.
Rigaud V, Zarka C, Kurian J, Harlamova D, Elia A, Kasatkin N, et al.
JCI Insight
. 2022 Jun;
7(15).
PMID: 35771638
Developmental cardiac tissue is regenerative while operating under low oxygen. After birth, ambient oxygen is associated with cardiomyocyte cell cycle exit and regeneration. Likewise, cardiac metabolism undergoes a shift with...
5.
Schena G, Murray E, Hildebrand A, Headrick A, Yang Y, Koch K, et al.
Am J Physiol Heart Circ Physiol
. 2021 Oct;
321(6):H1014-H1029.
PMID: 34623184
Heart failure is the one of the leading causes of death in the United States. Heart failure is a complex syndrome caused by numerous diseases, including severe myocardial infarction (MI)....
6.
Johnson J, Mohsin S, Houser S
Int J Mol Sci
. 2021 Aug;
22(15).
PMID: 34360531
Cardiac diseases such as myocardial infarction (MI) can lead to adverse remodeling and impaired contractility of the heart due to widespread cardiomyocyte death in the damaged area. Current therapies focus...
7.
Yang Y, Kurian J, Schena G, Johnson J, Kubo H, Travers J, et al.
Circulation
. 2021 Feb;
143(7):699-712.
PMID: 33587660
Background: The heart undergoes physiological hypertrophy during pregnancy in healthy individuals. Metabolic syndrome (MetS) is now prevalent in women of child-bearing age and might add risks of adverse cardiovascular events...
8.
Gross P, Johnson J, Romero C, Eaton D, Poulet C, Sanchez-Alonso J, et al.
Circ Res
. 2020 Oct;
128(1):92-114.
PMID: 33092464
Rationale: Ca-induced Ca release (CICR) in normal hearts requires close approximation of L-type calcium channels (LTCCs) within the transverse tubules (T-tubules) and RyR (ryanodine receptors) within the junctional sarcoplasmic reticulum....
9.
Harper S, Johnson J, Borghetti G, Zhao H, Wang T, Wallner M, et al.
Circ Res
. 2018 Dec;
123(11):1220-1231.
PMID: 30571461
Rationale: Possible beneficial effects of GDF11 (growth differentiation factor 11) on the normal, diseased, and aging heart have been reported, including reversing aging-induced hypertrophy. These effects have not been well...
10.
Pfleger J, Gross P, Johnson J, Carter R, Gao E, Tilley D, et al.
J Mol Cell Cardiol
. 2018 Sep;
123:108-117.
PMID: 30171848
Increased G protein-coupled receptor kinase (GRK)2 is central to heart failure (HF) pathogenesis, via desensitization of β-adrenergic receptors and loss of contractile reserve. Since GRK2 has been shown to compromise...