J P Mattson
Overview
Explore the profile of J P Mattson including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
6
Citations
67
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Mattson J, Poole D, Hahn S, Musch T, Hinkle R, Isfort R
Respir Physiol Neurobiol
. 2008 Mar;
161(2):119-24.
PMID: 18314399
Patients with chronic obstructive pulmonary disease (COPD) demonstrate a limited exercise capacity. It is unknown whether muscle fiber atrophy and subsequent decrease in force production contributes to this functional limitation....
2.
Mattson J, Delp M, Poole D
Eur Respir J
. 2004 Jun;
23(5):703-7.
PMID: 15176683
Patients afflicted with emphysema demonstrate altered peripheral skeletal muscle fibre composition and atrophy. It is unknown whether these alterations are general to all skeletal muscles independent of function, phenotype or...
3.
Mattson J, Ross C, Kilgore J, Musch T
Med Sci Sports Exerc
. 2000 Feb;
32(2):365-9.
PMID: 10694118
Purpose: The purpose of this investigation was to examine the relationship between the expression of HSP60 and GRP75 and the oxidative potential of skeletal muscle as assessed by the citrate...
4.
Mattson J, Poole D
J Appl Physiol (1985)
. 1998 Jul;
85(1):210-4.
PMID: 9655777
Skeletal muscle oxidative enzyme capacity is impaired in patients suffering from emphysema and chronic obstructive pulmonary disease. This effect may result as a consequence of the physiological derangements because of...
5.
Delp M, Duan C, Mattson J, Musch T
J Appl Physiol (1985)
. 1997 Oct;
83(4):1291-9.
PMID: 9338439
One of the primary consequences of left ventricular dysfunction (LVD) after myocardial infarction is a decrement in exercise capacity. Several factors have been hypothesized to account for this decrement, including...
6.
Pickar J, Mattson J, Lloyd S, Musch T
J Appl Physiol (1985)
. 1997 Jul;
83(1):323-7.
PMID: 9216979
Abnormalities intrinsic to skeletal muscle are thought to contribute to decrements in exercise capacity found in individuals with chronic heart failure (CHF). Na+-K+-adenosinetriphosphatase (the Na+ pump) is essential for maintaining...