J N Engel
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Explore the profile of J N Engel including associated specialties, affiliations and a list of published articles.
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33
Citations
1278
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Recent Articles
1.
Kim J, Chan C, Elwell C, Singer M, Dierks T, Lemjabbar-Alaoui H, et al.
Cell Microbiol
. 2013 Mar;
15(9):1560-71.
PMID: 23480519
The first step in attachment of Chlamydia to host cells is thought to involve reversible binding to host heparan sulfate proteoglycans (HSPGs), polymers of variably sulfated repeating disaccharide units coupled...
2.
Elwell C, Engel J
Cell Microbiol
. 2005 Apr;
7(5):725-39.
PMID: 15839901
Chlamydia spp. are major causes of important human diseases, but dissecting the host-pathogen interactions has been hampered by the lack of bacterial genetics and the difficulty in carrying out forward...
3.
Kierbel A, Gassama-Diagne A, Mostov K, Engel J
Mol Biol Cell
. 2005 Mar;
16(5):2577-85.
PMID: 15772151
Several Pseudomonas aeruginosa strains are internalized by epithelial cells in vitro and in vivo, but the host pathways usurped by the bacteria to enter nonphagocytic cells are not clearly understood....
4.
Gabel B, Elwell C, van Ijzendoorn S, Engel J
Infect Immun
. 2004 Nov;
72(12):7367-73.
PMID: 15557670
Using pharmacologic and biochemical criteria, we evaluated whether uptake of four different Chlamydia trachomatis serovars, D, E, K, and L2, was dependent upon lipid rafts. Our data suggest that lipid...
5.
Garrity-Ryan L, Shafikhani S, Balachandran P, Nguyen L, Oza J, Jakobsen T, et al.
Infect Immun
. 2003 Dec;
72(1):546-58.
PMID: 14688136
ExoT is a type III secreted effector protein found in almost all strains of Pseudomonas aeruginosa and is required for full virulence in an animal model of acute pneumonia. It...
6.
Kazmierczak B, Engel J
Infect Immun
. 2002 Mar;
70(4):2198-205.
PMID: 11895987
The Pseudomonas aeruginosa protein ExoT is a bacterial GTPase-activating protein (GAP) that has in vitro activity toward Rho, Rac, and Cdc42 GTPases. Expression of ExoT both inhibits the internalization of...
7.
Kazmierczak B, Mostov K, Engel J
Annu Rev Microbiol
. 2001 Sep;
55:407-35.
PMID: 11544362
Many pathogens must surmount an epithelial cell barrier in order to establish an infection. While much has been learned about the interaction of bacterial pathogens with cultured epithelial cells, the...
8.
Geiser T, Kazmierczak B, Matthay M, Engel J
Cell Microbiol
. 2001 Apr;
3(4):223-36.
PMID: 11298646
The nosocomial pathogen Pseudomonas aeruginosa causes clinical infection in the setting of pre-existing epithelial tissue damage, an association that is mirrored by the increased ability of P. aeruginosa to bind,...
9.
Kazmierczak B, Jou T, Mostov K, Engel J
Cell Microbiol
. 2001 Feb;
3(2):85-98.
PMID: 11207623
The Gram-negative pathogen Pseudomonas aeruginosa invades epithelial cells in vivo and in vitro. We have examined the pathway(s) by which epithelial cells internalize P. aeruginosa strain PA103 using Madin-Darby canine...
10.
van Ooij C, Kalman L, van Ijzendoorn , Nishijima M, Hanada K, Mostov K, et al.
Cell Microbiol
. 2001 Feb;
2(6):627-37.
PMID: 11207614
Chlamydia trachomatis, an important cause of human disease, is an obligate intracellular bacterial pathogen that relies on the eukaryotic host cell for its replication. Recent reports have revealed that the...