J Malcolm
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Explore the profile of J Malcolm including associated specialties, affiliations and a list of published articles.
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55
Citations
485
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Recent Articles
1.
Malcolm J, Nyirenda M, Brown J, Adrados-Planell A, Campbell L, Butcher J, et al.
J Autoimmun
. 2023 Jan;
135:102994.
PMID: 36706535
In rheumatoid arthritis, the emergence of anti-citrullinated autoimmunity is associated with HLA-antigen-T cell receptor complexes. The precise mechanisms underpinning this breach of tolerance are not well understood. Porphyromonas gingivalis expresses...
2.
Brown J, Campbell L, Malcolm J, Adrados Planell A, Butcher J, Culshaw S
J Dent Res
. 2018 Jun;
97(12):1399-1405.
PMID: 29928824
Innate lymphoid cells (ILCs) are a population of lymphocytes that act as the first line of immunologic defense at mucosal surfaces. The ILC family in the skin, lungs, and gastrointestinal...
3.
Simon-Soro A, Sherriff A, Sadique S, Ramage G, Macpherson L, Mira A, et al.
Sci Rep
. 2018 Jan;
8(1):1484.
PMID: 29367728
Understanding the triad of host response, microbiome and disease status is potentially informative for disease prediction, prevention, early intervention and treatment. Using longitudinal assessment of saliva and disease status, we...
4.
Ramage G, Lappin D, Millhouse E, Malcolm J, Jose A, Yang J, et al.
J Periodontal Res
. 2016 Jun;
52(3):325-333.
PMID: 27330034
Background And Objective: Different bacteria differentially stimulate epithelial cells. Biofilm composition and viability are likely to influence the epithelial response. In vitro model systems are commonly used to investigate periodontitis-associated...
5.
Malcolm J, Millington O, Millhouse E, Campbell L, Adrados Planell A, Butcher J, et al.
J Dent Res
. 2016 Mar;
95(6):704-10.
PMID: 26933137
Periodontitis is a chronic inflammatory and bone-destructive disease. Development of periodontitis is associated with dysbiosis of the microbial community, which may be caused by periodontal bacteria, such as Porphyromonas gingivalis...
6.
Campbell L, Millhouse E, Malcolm J, Culshaw S
Mol Oral Microbiol
. 2015 Oct;
31(6):445-456.
PMID: 26505640
The microbial plaque biofilm resides adjacent to the tissue-destructive inflammatory infiltrate in periodontitis. Although not sufficient, this biofilm is necessary for this inflammatory response. Patients with periodontitis generate antibodies specific...
7.
Alberga A, Prudhomme D, Kenny G, Goldfield G, Hadjiyannakis S, Gougeon R, et al.
Int J Obes (Lond)
. 2015 Jul;
39(10):1494-500.
PMID: 26202452
Objectives: To investigate the effects of aerobic training, resistance training, or both on abdominal subcutaneous fat (subcutaneous adipose tissue (SAT)) (deep and superficial), visceral fat (visceral adipose tissue (VAT)), apolipoproteins...
8.
Malcolm J, Awang R, Oliver-Bell J, Butcher J, Campbell L, Adrados Planell A, et al.
J Dent Res
. 2015 Mar;
94(7):968-75.
PMID: 25808546
Cytokines mediate the balance between protective and destructive immunity in periodontitis. We sought to investigate the role of IL-33 in periodontitis. The expression of IL-33 in gingival tissue from healthy...
9.
Oliver-Bell J, Butcher J, Malcolm J, MacLeod M, Adrados Planell A, Campbell L, et al.
Mol Oral Microbiol
. 2014 Sep;
30(2):160-9.
PMID: 25219939
Periodontitis (PD) results from complex interactions between a dysbiotic oral microbiota and a dysregulated host immune response. The inflammatory infiltrate in the gingiva of PD patients includes an abundance of...
10.
Malcolm J, Sherriff A, Lappin D, Ramage G, Conway D, Macpherson L, et al.
Mol Oral Microbiol
. 2014 Jun;
29(6):284-93.
PMID: 24890264
Secretion of antimicrobial proteins (AMPs) and salivary antibodies can modify biofilm formation at host body surfaces. In adolescents, associations have been reported between dental caries and salivary AMPs. AMPs demonstrate...