J J Grantham
Overview
Explore the profile of J J Grantham including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
160
Citations
2484
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Grantham J, Cook L, Torres V, Bost J, Chapman A, Harris P, et al.
Kidney Int
. 2007 Oct;
73(1):108-16.
PMID: 17960141
The Consortium of Radiologic Imaging Studies of Polycystic Kidney Disease (CRISP) recently showed that renal enlargement in autosomal-dominant polycystic kidney disease mimicked exponential growth. We determined the effects of cyst...
2.
Sutters M, Yamaguchi T, Maser R, Magenheimer B, St John P, Abrahamson D, et al.
Kidney Int
. 2001 Jul;
60(2):484-94.
PMID: 11473631
Background: Polycystic kidney disease (PKD) is characterized by the abnormal proliferation of tubular epithelial cells. It was recently shown that the growth of PKD cyst-lining cells is stimulated by cyclic...
3.
Grantham J
Curr Opin Nephrol Hypertens
. 2001 Jul;
10(4):533-42.
PMID: 11458035
Collated in this highly personal commentary are the most important research findings of the past 10 years that deal primarily with the renal manifestations of inherited polycystic kidney diseases. Progress...
4.
Wallace D, Rome L, SULLIVAN L, Grantham J
Am J Physiol Renal Physiol
. 2001 May;
280(6):F1019-29.
PMID: 11352842
We used an unambiguous in vitro method to determine if inner medullary collecting ducts (IMCD) have intrinsic capacities to absorb and secrete solutes and fluid in an isotonic medium. IMCD(1),...
5.
CALVET J, Grantham J
Semin Nephrol
. 2001 Mar;
21(2):107-23.
PMID: 11245774
Autosomal dominant polycystic kidney disease (ADPKD) is a major, inherited disorder that is characterized by the growth of large, fluid-filled cysts from the tubules and collecting ducts of affected kidneys,...
6.
Grantham J, CALVET J
Proc Natl Acad Sci U S A
. 2001 Feb;
98(3):790-2.
PMID: 11158545
No abstract available.
7.
Sise C, Kusaka M, Wetzel L, Winklhofer F, Cowley B, Cook L, et al.
Kidney Int
. 2000 Dec;
58(6):2492-501.
PMID: 11115083
Background: Autosomal dominant polycystic kidney disease (ADPKD) is characterized by progressive renal enlargement and renal failure. We evaluated sequential radiocontrast-enhanced computed tomography (CT) scans to determine the rate of kidney...
8.
Yamaguchi T, Pelling J, Ramaswamy N, Eppler J, Wallace D, Nagao S, et al.
Kidney Int
. 2000 Apr;
57(4):1460-71.
PMID: 10760082
Background: : Cellular proliferation is a key factor in the enlargement of renal cysts in autosomal dominant polycystic kidney disease (ADPKD). We determined the extent to which adenosine 3':5'-cyclic monophosphate...
9.
Wallace D, Tomich J, Eppler J, Iwamoto T, Grantham J, SULLIVAN L
Biochim Biophys Acta
. 2000 Mar;
1464(1):69-82.
PMID: 10704921
A synthetic Cl(-) channel-forming peptide, C-K4-M2GlyR, applied to the apical membrane of human epithelial cell monolayers induces transepithelial Cl(-) and fluid secretion. The sequence of the core peptide, M2GlyR, corresponds...
10.
Nagao S, Yamaguchi T, Kasahara M, Kusaka M, Matsuda J, Ogiso N, et al.
Am J Kidney Dis
. 2000 Feb;
35(2):221-6.
PMID: 10676720
Epithelial proliferation, extracellular matrix remodeling, and interstitial inflammation are central elements in the pathogenesis of slowly progressive polycystic kidney disorders. Probucol, an antioxidant that lowers plasma cholesterol, has been shown...