J E Springer
Overview
Explore the profile of J E Springer including associated specialties, affiliations and a list of published articles.
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Articles
52
Citations
1336
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Recent Articles
1.
McKechnie T, Wang J, Springer J, Gross P, Forbes S, Eskicioglu C
Colorectal Dis
. 2019 Sep;
22(6):663-678.
PMID: 31490000
Aim: Patients with inflammatory bowel disease (IBD) are at increased risk of postoperative venous thromboembolism (VTE) following major abdominal surgery. The pathogenesis is multifactorial and not fully understood. A combination...
2.
MINDEN P, Springer J
J Am Med Assoc
. 2010 Mar;
134(13):1061-4.
PMID: 20251987
No abstract available.
3.
Mbye L, Singh I, Sullivan P, Springer J, Hall E
Exp Neurol
. 2007 Nov;
209(1):243-53.
PMID: 18022160
Following traumatic brain injury (TBI), mitochondrial function becomes compromised. Mitochondrial dysfunction is characterized by intra-mitochondrial Ca(2+) accumulation, induction of oxidative damage, and mitochondrial permeability transition (mPT). Experimental studies show that...
4.
Sullivan P, Rabchevsky A, Waldmeier P, Springer J
J Neurosci Res
. 2004 Dec;
79(1-2):231-9.
PMID: 15573402
Experimental traumatic brain injury (TBI) and spinal cord injury (SCI) result in a rapid and significant necrosis of neuronal tissue at the site of injury. In the ensuing hours and...
5.
Zwick M, Teng L, Mu X, Springer J, Davis B
Exp Neurol
. 2001 Sep;
171(2):342-50.
PMID: 11573987
This study examined the role of glial cell line-derived neurotrophic factor (GDNF) in synaptic plasticity at the developing neuromuscular junction. Transgenic mice overexpressing GDNF in skeletal muscle under the myosin...
6.
Springer J, Nottingham S, McEwen M, Azbill R, Jin Y
Clin Chem Lab Med
. 2001 Jun;
39(4):299-307.
PMID: 11388652
Apoptotic cell death is a fundamental and highly regulated biological process in which a cell is instructed to participate actively in its own demise. This process of cellular suicide is...
7.
Mu X, Azbill R, Springer J
J Neurotrauma
. 2000 Sep;
17(9):773-80.
PMID: 11011817
The potential use of riluzole (a glutamate release inhibitor) alone or in combination with methyl-prednisolone (MP) in treating acute spinal cored injury (SCI) was examined. Rats received a contusion injury...
8.
Springer J, Azbill R, Nottingham S, Kennedy S
J Neurosci
. 2000 Sep;
20(19):7246-51.
PMID: 11007881
We report here that activation of the caspase-3 apoptotic cascade in spinal cord injury is regulated, in part, by calcineurin-mediated BAD dephosphorylation. BAD, a proapoptotic member of the bcl-2 gene...
9.
Azbill R, Mu X, Springer J
Brain Res
. 2000 Jul;
871(2):175-80.
PMID: 10899284
The purpose of this study was to examine the effect of the anti-convulsant agent, riluzole, on high-affinity glutamate uptake as measured in rat spinal cord synaptosomes. The rate of glutamate...
10.
Mu X, Azbill R, Springer J
Brain Res
. 2000 Jun;
870(1-2):66-72.
PMID: 10869502
Rats received a contusion injury to the spinal cord followed by treatment with riluzole (a glutamate release inhibitor, 8 mg/kg), methylprednisolone (MP 30 mg/kg) or both. At 4 h following...