J E Franck
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Explore the profile of J E Franck including associated specialties, affiliations and a list of published articles.
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18
Citations
225
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Recent Articles
1.
Westenbroek R, Bausch S, Lin R, Franck J, Noebels J, Catterall W
J Neurosci
. 1998 Apr;
18(7):2321-34.
PMID: 9502793
Anti-peptide antibodies that specifically recognize the alpha1 subunit of class A-D voltage-gated Ca2+ channels and a monoclonal antibody (MANC-1) to the alpha2 subunit of L-type Ca2+ channels were used to...
2.
Physiologic and morphologic characteristics of granule cell circuitry in human epileptic hippocampus
Franck J, Pokorny J, Kunkel D, Schwartzkroin P
Epilepsia
. 1995 Jun;
36(6):543-58.
PMID: 7555966
Morphological and electrophysiological techniques were used to examine granule cells and their mossy fiber axons in nine surgically resected hippocampal specimens from temporal lobe epilepsy (TLE) patients. Timm histochemistry showed...
3.
Hampson D, Theriault E, Huang X, Kristensen P, Pickering D, Franck J, et al.
Neuroscience
. 1994 May;
60(2):325-36.
PMID: 8072687
Amplification of complementary DNA by the polymerase chain reaction and anti-peptide antibodies were used to characterize the expression of two alternatively spliced forms of a metabotropic glutamate receptor (mGluR1 alpha...
4.
Anderson W, Franck J, Stahl W, Maki A
Epilepsy Res
. 1994 Mar;
17(3):221-31.
PMID: 8013444
The effects of intraventricular injection of kainic acid on the Na,K-ATPase (Na,K pump) were examined in discrete pyramidal cell regions of rat hippocampus. [3H]Ouabain binding was used to quantitate Na,K-ATPase...
5.
Haglund M, Berger M, Kunkel D, Franck J, Ghatan S, Ojemann G
J Neurosurg
. 1992 Aug;
77(2):209-16.
PMID: 1352540
The role of specific neuronal populations in epileptic foci was studied by comparing epileptic and non-epileptic cortex removed from patients with low-grade gliomas. Epileptic and nearby (within 1 to 2...
6.
Nakajima S, Franck J, Bilkey D, Schwartzkroin P
Hippocampus
. 1991 Jan;
1(1):67-78.
PMID: 1669343
Following kainate (KA)-induced lesions of subfield CA3--a lesion relevant to human temporal lobe epilepsy--remaining pyramidal cells in CA1 display synchronous hyperexcitability associated with a loss of synaptic inhibition. Despite this...
7.
Franck J, Roberts D
Neurosci Lett
. 1990 Oct;
118(2):159-63.
PMID: 2274264
In the hippocampi of individuals with temporal lobe epilepsy, cells in CA1, CA3, and the dentate hilus are often gone, but dentate granule cells and CA2 are relatively spared. Several...
8.
Newell D, Malouf A, Franck J
Neurosci Lett
. 1990 Aug;
116(3):325-30.
PMID: 1978744
Ischemic damage to the brain, whether induced experimentally or observed clinically, often produces a pattern of delayed selective cell death in subfield CA1 of hippocampus which has been associated with...
9.
Franck J, Ginter K, Schwartzkroin P
Epilepsia
. 1989 Jan;
30(1):1-6.
PMID: 2912711
Development of clonic-tonic flurothyl-induced seizures was examined in both normal and genetically epilepsy-prone rats (GEPRs). At each age, from 10 to 30 days, clonus occurred at significantly shorter latencies in...
10.
Franck J, Kunkel D, Baskin D, Schwartzkroin P
J Neurosci
. 1988 Jun;
8(6):1991-2002.
PMID: 2838591
Following kainate lesions of hippocampal subfield CA3, the remaining CA 1 pyramidal cells become hyperexcitable. This lesion is of interest because, morphologically, it resembles the damage often seen in cases...