J C Quindry
Overview
Explore the profile of J C Quindry including associated specialties, affiliations and a list of published articles.
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Articles
6
Citations
67
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0
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Recent Articles
1.
Spaulding H, Quindry T, Quindry J, Selsby J
Exp Physiol
. 2020 Apr;
105(6):989-999.
PMID: 32267561
New Findings: What is the central question of this study? We previously demonstrated that quercetin transiently preserved respiratory function in dystrophin-deficient mice. To gain lasting therapeutic benefits, we tested quercetin...
2.
Spaulding H, Ballmann C, Quindry J, Hudson M, Selsby J
JRSM Cardiovasc Dis
. 2019 Oct;
8:2048004019879581.
PMID: 31656622
Background: Duchenne muscular dystrophy is a muscle wasting disease caused by dystrophin gene mutations resulting in dysfunctional dystrophin protein. Autophagy, a proteolytic process, is impaired in dystrophic skeletal muscle though...
3.
Spaulding H, Kelly E, Quindry J, Sheffield J, Hudson M, Selsby J
Acta Physiol (Oxf)
. 2017 Aug;
222(2).
PMID: 28834378
Aim: Duchenne muscular dystrophy is caused by the absence of functional dystrophin protein and results in a host of secondary effects. Emerging evidence suggests that dystrophic pathology includes decreased pro-autophagic...
4.
Borst S, Quindry J, Yarrow J, Conover C, Powers S
Horm Metab Res
. 2009 Oct;
42(2):122-9.
PMID: 19862668
We tested the hypothesis that chronic testosterone treatment would promote a cardioprotective phenotype against ischemia/reperfusion (I/R) injury. For this study, 3-month-old F344 male rats underwent sham-surgery, orchiectomy (ORX), or ORX...
5.
McAnulty S, Hosick P, McAnulty L, Quindry J, Still L, Hudson M, et al.
Appl Physiol Nutr Metab
. 2007 Dec;
32(6):1148-55.
PMID: 18059589
Urate is a metabolic end product of purine metabolism that contributes about 66% of the antioxidant capacity of plasma. The objective of this study was to evaluate the importance of...
6.
Lennon S, Quindry J, French J, Kim S, Mehta J, Powers S
Acta Physiol Scand
. 2004 Sep;
182(2):161-9.
PMID: 15450112
Unlabelled: It is well established that both short-term (1-5 days) and long-term (weeks to months) high intensity exercise (i.e. 70-75%VO2max) provides cardioprotection against ischaemia-reperfusion injury. However, it is unclear if...