I Anan
Overview
Explore the profile of I Anan including associated specialties, affiliations and a list of published articles.
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14
Citations
81
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0
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Recent Articles
1.
Wixner J, Tornblom H, Karling P, Anan I, Lindberg G
Neurogastroenterol Motil
. 2018 Apr;
30(9):e13354.
PMID: 29655299
Background: Gastrointestinal complications are common in hereditary transthyretin amyloid (ATTRm) amyloidosis. The underlying mechanisms have not been fully elucidated, and the patients' small bowel function remains largely unexplored. The aim...
2.
Wixner J, Karling P, Rydh A, Hornsten R, Wiklund U, Anan I, et al.
Neurogastroenterol Motil
. 2012 Aug;
24(12):1111-e568.
PMID: 22897426
Background: Gastrointestinal (GI) complications are common in hereditary transthyretin amyloidosis and an autonomic dysfunction has been considered to explain these symptoms. The aim of this study was to investigate the...
3.
Suhr O, Anan I, Backman C, Karlsson A, Lindqvist P, Morner S, et al.
J Intern Med
. 2007 Dec;
263(3):294-301.
PMID: 18069997
Objectives: Cardiomyopathy is a well known complication in familial amyloidotic polyneuropathy (FAP). Troponin T and B-natriuretic peptide (BNP) have been shown to be excellent markers for heart complications in AL-amyloidosis....
4.
Matsunaga N, Anan I, Rosenberg P, Nagai R, Lundstrom O, Horiuchi S, et al.
Scand J Clin Lab Invest
. 2005 Aug;
65(4):263-71.
PMID: 16076681
Background: Kidney failure is a common complication in familial amyloidotic polyneuropathy (FAP). It has been suggested that advanced glycation end products (AGEs) play an important role in the development and...
5.
Nyhlin N, Anan I, El S, Ando Y, Suhr O
J Intern Med
. 2002 Mar;
251(2):136-41.
PMID: 11905589
Objectives: Liver transplantation halt the progress of familial amyloidotic polyneuropathy (FAP). Oxidative stress has been implicated in amyloid toxicity and formation. The objective of this study was to establish whether...
6.
Anan I, El-Salhy M, Ando Y, Terazaki H, Suhr O
Acta Neuropathol
. 2001 Oct;
102(3):227-32.
PMID: 11585246
Gastrointestinal (GI) complications in familial amyloidotic polyneuropathy (FAP) are invariably present during the course of the disease. The aim of this study was to investigate amyloid deposits in the myenteric...
7.
Suhr O, Lang K, Wikstrom L, Anan I, Ando Y, El-Salhy M, et al.
Scand J Clin Lab Invest
. 2001 Apr;
61(1):11-8.
PMID: 11300606
Since oxidative stress has been implicated in amyloid diseases, a study of scavenger treatment of hereditary transthyretin amyloidosis was undertaken on 23 familial amyloidotic polyneuropathy (FAP) patients. Nine patients had...
8.
Anan I, El-Salhy M, Nyhlin N, Suhr O
Transplantation
. 2000 Sep;
70(5):794-9.
PMID: 11003360
Background: The aim of this study was to investigate familial amyloidotic polyneuropathy, Portuguese type patients' endocrine cell content in the stomach and duodenum before and after liver transplantation, and to...
9.
Janunger T, Anan I, Holmgren G, Lovheim O, Ohlsson P, Suhr O, et al.
Amyloid
. 2000 Jun;
7(2):137-40.
PMID: 10842718
Cardiac failure in transthyretin (TTR) amyloidosis patients has been shown to be caused by different mutations in the TTR gene. In the present case, a 73-year-old man from Northern Sweden...
10.
Mambule C, Ando Y, Anan I, Holmgren G, Sandgren O, Stigbrandt T, et al.
Biochim Biophys Acta
. 2000 Apr;
1474(3):331-6.
PMID: 10779684
The mechanism behind amyloid formation is unknown in all types of amyloidosis. Several substances can enhance amyloid formation in animal experiments. To induce secondary systemic amyloid (AA-type amyloid) formation, we...