Hisazumi Araki
Overview
Explore the profile of Hisazumi Araki including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
29
Citations
722
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Ono S, Kume S, Yasuda-Yamahara M, Yamahara K, Takeda N, Chin-Kanasaki M, et al.
Nephrol Dial Transplant
. 2017 Mar;
32(9):1477-1487.
PMID: 28339907
Background: O-linked β- N -acetylglucosamine modification O-GlcNAcylation) is a post-translational modification of intracellular proteins, serving as a nutrient sensor. Growing evidence has demonstrated its physiological and pathological importance in various...
2.
Kume S, Kondo M, Maeda S, Nishio Y, Yanagimachi T, Fujita Y, et al.
EBioMedicine
. 2016 Dec;
13:168-180.
PMID: 28005533
Glucose-stimulated insulin secretion (GSIS) by pancreatic β cells is biphasic. However, the physiological significance of biphasic GSIS and its relationship to diabetes are not yet fully understood. This study demonstrated...
3.
Iwai T, Kume S, Chin-Kanasaki M, Kuwagata S, Araki H, Takeda N, et al.
Int J Mol Sci
. 2016 Nov;
17(11).
PMID: 27834856
Saturated fatty acid (SFA)-related lipotoxicity is a pathogenesis of diabetes-related renal proximal tubular epithelial cell (PTEC) damage, closely associated with a progressive decline in renal function. This study was designed...
4.
Kuwagata S, Kume S, Chin-Kanasaki M, Araki H, Araki S, Nakazawa J, et al.
Kidney Int
. 2016 Sep;
90(6):1211-1225.
PMID: 27591086
Hypoxia causes proximal tubular cell damage in diabetes, even though proximal tubular cells have an adaptive system to combat hypoxia involving induction of hypoxia factor-1 (HIF-1) and inhibition of mechanistic...
5.
Tanaka Y, Kume S, Chin-Kanasaki M, Araki H, Araki S, Ugi S, et al.
Biochem Biophys Res Commun
. 2016 Jan;
470(3):539-545.
PMID: 26802469
Dipeptidyl peptidase (DPP)-4 inhibitors, a new class of antidiabetic agent, have recently been suggested to exert pleiotropic effects beyond glucose lowering. Renal prognosis in patients with diabetic nephropathy depends on...
6.
Takagi A, Kume S, Kondo M, Nakazawa J, Chin-Kanasaki M, Araki H, et al.
Sci Rep
. 2016 Jan;
6:18944.
PMID: 26732653
Autophagy is an intracellular degradation system activated, across species, by starvation. Although accumulating evidence has shown that mammalian autophagy is involved in pathogenesis of several modern diseases, its physiological role...
7.
Araki S, Haneda M, Koya D, Kondo K, Tanaka S, Arima H, et al.
Clin J Am Soc Nephrol
. 2015 Nov;
10(12):2152-8.
PMID: 26563378
Background And Objectives: We investigated the association of urinary potassium and sodium excretion with the incidence of renal failure and cardiovascular disease in patients with type 2 diabetes. Design, Setting,...
8.
Tanaka Y, Kume S, Araki H, Nakazawa J, Chin-Kanasaki M, Araki S, et al.
Free Radic Biol Med
. 2015 Oct;
89:831-41.
PMID: 26482866
Free fatty acid-bound albumin (FFA-albumin)-related oxidative stress is involved in the pathogenesis of proximal tubular cell (PTC) damage and subsequent renal dysfunction in patients with refractory proteinuria. Nicotinamide adenine dinucleotide...
9.
Tagawa A, Yasuda M, Kume S, Yamahara K, Nakazawa J, Chin-Kanasaki M, et al.
Diabetes
. 2015 Sep;
65(3):755-67.
PMID: 26384385
Overcoming refractory massive proteinuria remains a clinical and research issue in diabetic nephropathy. This study was designed to investigate the pathogenesis of massive proteinuria in diabetic nephropathy, with a special...
10.
Araki H, Ono S, Nishizawa Y, Deji N, Nakazawa J, Morita Y, et al.
Intern Med
. 2015 Sep;
54(17):2213-7.
PMID: 26328649
We report a case involving a 43-year-old Japanese woman with steroid-resistant focal segmental glomerular sclerosis (FSGS) and severe renal dysfunction, which was ameliorated by low-density lipoprotein apheresis (LDL-A). She had...