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H Cao-Danh

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Articles 16
Citations 143
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Recent Articles
1.
Friehs I, Margossian R, Moran A, Cao-Danh H, Moses M, Del Nido P
Basic Res Cardiol . 2005 Dec; 101(3):204-13. PMID: 16369727
Objective: Pressure-overload hypertrophy is associated with decreased capillary density in myocardium resulting in impaired substrate delivery. Treatment of hypertrophied hearts with vascular endothelial growth factor (VEGF) induces angiogenesis. Since angiogenesis...
2.
Friehs I, Stamm C, Cao-Danh H, McGowan F, Del Nido P
Ann Thorac Surg . 2001 Nov; 72(5):1650-6. PMID: 11722060
Background: Severe myocardial hypertrophy is associated with decreased tolerance to ischemia compared with normal hearts. We hypothesized that treatment with insulin-like growth factor-1 (IGF-1) improves postischemic myocardial recovery by increasing...
3.
Stamm C, Friehs I, Cowan D, Moran A, Cao-Danh H, Duebener L, et al.
Circulation . 2001 Sep; 104(12 Suppl 1):I350-5. PMID: 11568081
Background: Tumor necrosis factor (TNF)-alpha has been implicated in the pathogenesis of heart failure and ischemia-reperfusion injury. Effects of TNF-alpha are initiated by membrane receptors coupled to sphingomyelinase signaling and...
4.
Stamm C, Friehs I, Cowan D, Cao-Danh H, Noria S, Munakata M, et al.
Cardiovasc Res . 2001 Jun; 51(1):108-21. PMID: 11399253
Objective: Protein kinase C (PKC) activation impairs contractility in the normal heart but is protective during myocardial ischemia. We hypothesized that PKC remains activated post-ischemia and modulates myocardial excitation-contraction coupling...
5.
Friehs I, Moran A, Stamm C, Colan S, Takeuchi K, Cao-Danh H, et al.
Circulation . 1999 Nov; 100(19 Suppl):II187-93. PMID: 10567302
Background: Severe hypertrophy and heart failure are important risk factors in cardiac surgery. Early adaptive changes in hypertrophy include increased ventricular mass-to-cavity volume ratio (M/V ratio) and increased dependence on...
6.
Takeuchi K, Cao-Danh H, Kawai A, Ohkado A, Konishi H, McGowan F, et al.
Ann Thorac Surg . 1999 Oct; 68(3):903-7. PMID: 10509981
Background: Prolonged ischemia and inadequate myocardial preservation remain significant perioperative risk factors in cardiac transplantation. Long-term preservation techniques that have been effective in small rodent hearts have not been as...
7.
Takeuchi K, Del Nido P, Ibrahim A, Poutias D, Glynn P, Cao-Danh H, et al.
Surgery . 1999 Aug; 126(2):231-8. PMID: 10455889
Background: Mechanisms of cardiac dysfunction during endotoxemia are multiple and their targets uncertain. This study tested the hypothesis that endotoxin (LPS) induces abnormal calcium-activated contractile force in the heart. Methods:...
8.
Takeuchi K, Del Nido P, Ibrahim A, Cao-Danh H, Friehs I, Glynn P, et al.
J Thorac Cardiovasc Surg . 1999 Jan; 117(2):375-82. PMID: 9918980
Objective: The systemic inflammatory response is an important cause of organ dysfunction. The present study tested the hypothesis that 2 clinically used agents, amrinone and vesnarinone, would decrease inflammation and...
9.
Takeuchi K, McGowan Jr F, Glynn P, Moran A, Rader C, Cao-Danh H, et al.
Circulation . 1998 Dec; 98(19 Suppl):II234-9; discussion II240-1. PMID: 9852908
Background: Achieving successful myocardial preservation of hypertrophied hearts remains a difficult problem. Despite reportedly higher glycolytic potential, we have achieved limited benefit in hypertrophied hearts with strategies that successfully promote...
10.
Takeuchi K, Cao-Danh H, Friehs I, Glynn P, DAgostino D, Simplaceanu E, et al.
J Thorac Cardiovasc Surg . 1998 Aug; 116(2):335-43. PMID: 9699588
Objectives: Fructose-1,6-diphosphate is a glycolytic intermediate that has been shown experimentally to cross the cell membrane and lead to increased glycolytic flux. Because glycolysis is an important energy source for...