Gustavo J J Silva
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Explore the profile of Gustavo J J Silva including associated specialties, affiliations and a list of published articles.
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19
Citations
385
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Recent Articles
1.
Frisk M, Le C, Shen X, Roe A, Hou Y, Manfra O, et al.
J Am Coll Cardiol
. 2021 Jan;
77(4):405-419.
PMID: 33509397
Background: Whereas heart failure with reduced ejection fraction (HFrEF) is associated with ventricular dilation and markedly reduced systolic function, heart failure with preserved ejection fraction (HFpEF) patients exhibit concentric hypertrophy...
2.
Souza R, Alves C, Medeiros A, Rolim N, Silva G, Moreira J, et al.
Sci Rep
. 2018 Dec;
8(1):17772.
PMID: 30538258
Given the association between high aerobic capacity and the prevention of metabolic diseases, elucidating the mechanisms by which high aerobic capacity regulates whole-body metabolic homeostasis is a major research challenge....
3.
Silva G, Bye A, Azzouzi H, Wisloff U
Prog Cardiovasc Dis
. 2017 Jul;
60(1):130-151.
PMID: 28666746
A significant body of evidence supports the protective role of exercise training (ET) in cardiovascular diseases, skeletal muscle dystrophies, several types of cancer, Alzheimer disease or even in the recovery...
4.
Bye A, Rosjo H, Nauman J, Silva G, Follestad T, Omland T, et al.
J Mol Cell Cardiol
. 2016 May;
97:162-8.
PMID: 27192016
Coronary heart disease is the most common cause of death, and the number of individuals at risk is increasing. To better manage this pandemic, improved tool for risk prediction, including...
5.
Cannon M, Sillje H, Sijbesma J, Vreeswijk-Baudoin I, Ciapaite J, van der Sluis B, et al.
EMBO Mol Med
. 2015 Jul;
7(9):1229-43.
PMID: 26160456
Pathological cardiac hypertrophy is characterized by a shift in metabolic substrate utilization from fatty acids to glucose, but the molecular events underlying the metabolic remodeling remain poorly understood. Here, we...
6.
Bowen T, Rolim N, Fischer T, Baekkerud F, Medeiros A, Werner S, et al.
Eur J Heart Fail
. 2015 Feb;
17(3):263-72.
PMID: 25655080
Aims: Peripheral muscle dysfunction is a key mechanism contributing to exercise intolerance (i.e. breathlessness and fatigue) in heart failure patients with preserved ejection fraction (HFpEF); however, the underlying molecular and...
7.
Bajgelman M, Dos Santos L, Silva G, Nakamuta J, Sirvente R, Chaves M, et al.
Virology
. 2014 Dec;
476:106-114.
PMID: 25543961
Here we present the application of our adeno-associated virus (AAV2) vector where transgene expression is driven by a synthetic, p53-responsive promoter, termed PG, used to supply human vascular endothelial growth...
8.
Miana L, Assad R, Abduch M, Silva G, Nogueira A, Aiello V, et al.
Ann Thorac Surg
. 2013 Feb;
95(4):1422-8.
PMID: 23434258
Background: Traditional pulmonary artery banding (PAB) is not always suitable for mature subpulmonary ventricle retraining. We sought to assess in detail the myocardial morphologic adaptations of two different protocols for...
9.
Favaro G, Assad R, Abduch M, Silva G, Gomes G, Andrade J, et al.
J Thorac Cardiovasc Surg
. 2012 Aug;
145(5):1345-1351.e4.
PMID: 22925567
Background: Ventricle retraining with abrupt systolic overload can cause myocardial edema and necrosis, followed by late ventricular failure. Intermittent systolic overload could minimize the inadequacy of conventional pulmonary artery banding....
10.
Assad R, Atik F, Oliveira F, Fonseca-Alaniz M, Abduch M, Silva G, et al.
J Thorac Cardiovasc Surg
. 2011 Sep;
142(5):1108-13, 1113.e1.
PMID: 21907360
Objective: Increased myocardial glucose-6-phosphate dehydrogenase (G6PD) activity occurs in heart failure. This study compared G6PD activity in 2 protocols of right ventricle (RV) systolic overload in young goats. Methods: Twenty-seven...