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Greg Thill

Explore the profile of Greg Thill including associated specialties, affiliations and a list of published articles. Areas
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Articles 6
Citations 832
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Recent Articles
1.
Tummala S, Titus M, Wilson L, Wang C, Ciatto C, Thill G, et al.
Biotechnol Prog . 2012 Nov; 29(2):415-24. PMID: 23172735
Traditional metabolic engineering approaches, including homologous recombination, zinc-finger nucleases, and short hairpin RNA, have previously been used to generate biologics with specific characteristics that improve efficacy, potency, and safety. An...
2.
Mi S, Miller R, Lee X, Scott M, Shulag-Morskaya S, Shao Z, et al.
Nat Neurosci . 2005 May; 8(6):745-51. PMID: 15895088
The control of myelination by oligodendrocytes in the CNS is poorly understood. Here we show that LINGO-1 is an important negative regulator of this critical process. LINGO-1 is expressed in...
3.
Shao Z, Browning J, Lee X, Scott M, Shulga-Morskaya S, Allaire N, et al.
Neuron . 2005 Feb; 45(3):353-9. PMID: 15694322
Myelin-associated inhibitory factors (MAIFs) are inhibitors of CNS axonal regeneration following injury. The Nogo receptor complex, composed of the Nogo-66 receptor 1 (NgR1), neurotrophin p75 receptor (p75), and LINGO-1, represses...
4.
Mathur P, Murray B, Crowell T, Gardner H, Allaire N, Hsu Y, et al.
Genomics . 2004 Jun; 83(6):1151-63. PMID: 15177568
Peptidoglycan recognition proteins (PGRPs or PGLYRPs) are pattern recognition molecules that are found in insects and mammals and are critical for innate immune responses. PGRPs bind peptidoglycan, a ubiquitous component...
5.
Mi S, Lee X, Shao Z, Thill G, Ji B, Relton J, et al.
Nat Neurosci . 2004 Feb; 7(3):221-8. PMID: 14966521
Axon regeneration in the adult CNS is prevented by inhibitors in myelin. These inhibitors seem to modulate RhoA activity by binding to a receptor complex comprising a ligand-binding subunit (the...
6.
Gorelik L, Cutler A, Thill G, Miklasz S, Shea D, Ambrose C, et al.
J Immunol . 2004 Jan; 172(2):762-6. PMID: 14707045
Herein we demonstrate that B cell-activating factor of the TNF family (BAFF), a B cell survival factor, also regulates CD21/35 and CD23 expression. BAFF blockade in wild-type mice down-modulates CD21/35...